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"content": "\u003cp>Can't cool off this summer? Heat waves can slow us down in ways we may not realize.[contextly_sidebar id=\"vfD9z3obGh2C18mIRbWsth99DmsY1OtQ\"]\u003c/p>\n\u003cp>New research suggests heat stress can muddle our thinking, making simple math a little harder to do.\u003c/p>\n\u003cp>\"There's evidence that our brains are susceptible to temperature abnormalities,\" says \u003ca href=\"https://www.hsph.harvard.edu/joseph-allen/\" target=\"_blank\" rel=\"noopener\">Joe Allen\u003c/a>, co-director of the \u003ca href=\"https://www.hsph.harvard.edu/c-change/\" target=\"_blank\" rel=\"noopener\">Center for Climate, Health and the Global Environment\u003c/a> at Harvard University. And as the climate changes, temperatures spike and \u003ca href=\"https://science2017.globalchange.gov/chapter/6/\" target=\"_blank\" rel=\"noopener\">heat waves are more frequent\u003c/a>.\u003c/p>\n\u003cp>To learn more about how the heat influences young, healthy adults, Allen and his colleagues studied college students living in dorms during a summer heat wave in Boston.\u003c/p>\n\u003cp>Half of the students lived in buildings with central AC, where the indoor air temperature averaged 71 degrees. The other half lived in dorms with no AC, where air temperatures averaged almost 80 degrees.\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>\"In the morning, when they woke up, we pushed tests out to their cellphones,\" explains Allen. The students took two tests a day for 12 consecutive days.\u003c/p>\n\u003cp>One test, which included basic addition and subtraction, measured cognitive speed and memory. A second test assessed attention and processing speed.\u003c/p>\n\u003cp>\"We found that the students who were in the non-air-conditioned buildings actually had slower reaction times: 13 percent lower performance on basic arithmetic tests, and nearly a 10 percent reduction in the number of correct responses per minute,\" Allen explains.\u003c/p>\n\u003cp>The results, \u003ca href=\"http://journals.plos.org/plosmedicine/article?id=10.1371/journal.pmed.1002605\" target=\"_blank\" rel=\"noopener\">published in PLOS Medicine\u003c/a>, may come as a surprise. \"I think it's a little bit akin to the frog in the boiling water,\" Allen says. There's a \"slow, steady — largely imperceptible — rise in temperature, and you don't realize it's having an impact on you.\"\u003c/p>\n\u003cp>The findings add to a growing body of evidence that documents the effect of heat on mental performance, both in schools and workplaces.\u003c/p>\n\u003cp>For instance, a 2006 study from researchers at the Lawrence Berkeley National Lab found that when office temperatures rise above the mid-70s, \u003ca href=\"https://indoor.lbl.gov/publications/effect-temperature-task-performance\" target=\"_blank\" rel=\"noopener\">workers' performance begins to drop off\u003c/a>. Researchers reviewed multiple studies that evaluated performance on common office tasks\u003cem>.\u003c/em> The study found that worker productivity is highest at about 72 degrees. When temperatures exceeded the mid-80s, worker productivity decreased by about 9 percent.\u003c/p>\n\u003cp>Another, \u003ca href=\"https://www.sciencedirect.com/science/article/pii/S0360132316304723\" target=\"_blank\" rel=\"noopener\">more recent study\u003c/a> compared worker performance in green-certified buildings and typical office buildings. They found a dip in cognitive function linked to conditions in the indoor environment, including higher indoor temperatures and poor lighting.\u003c/p>\n\u003cp>And, when it comes to performance in the classroom, a study funded by the Harvard Environmental Economics Program finds that taking a standardized test on a very hot day is\u003ca href=\"https://scholar.harvard.edu/files/jisungpark/files/paper_nyc_aejep.pdf\" target=\"_blank\" rel=\"noopener\"> linked to poorer performance\u003c/a>. The study includes an analysis of test scores from students in New York City who take a series of high-school exams called the Regents Exams.\u003c/p>\n\u003cp>The author, R. Jisung Park, assistant professor at the University of California, Los Angeles, writes that compared with a 72-degree day, \"taking an exam on a 90◦F day leads to a 10.9 percent lower likelihood of passing a particular subject (e.g. Algebra), which in turn affects probability of graduation.\"\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>There's still a lot to learn about how our brains and bodies respond to heat. \"We all tend to think we can compensate, we can do just fine\" during heat waves says Allen. But he says the \"evidence shows that the indoor temperature can have a dramatic impact on our ability to be productive and learn.\"\u003c/p>\n\u003cdiv class=\"fullattribution\">Copyright 2018 NPR. To see more, visit http://www.npr.org/.\u003cimg src=\"https://www.google-analytics.com/__utm.gif?utmac=UA-5828686-4&utmdt=Heat+Making+You+Lethargic%3F+Research+Shows+It+Can+Slow+Your+Brain%2C+Too&utme=8(APIKey)9(MDAxOTAwOTE4MDEyMTkxMDAzNjczZDljZA004)\">\u003c/div>\n\n",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp>Can't cool off this summer? Heat waves can slow us down in ways we may not realize.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>New research suggests heat stress can muddle our thinking, making simple math a little harder to do.\u003c/p>\n\u003cp>\"There's evidence that our brains are susceptible to temperature abnormalities,\" says \u003ca href=\"https://www.hsph.harvard.edu/joseph-allen/\" target=\"_blank\" rel=\"noopener\">Joe Allen\u003c/a>, co-director of the \u003ca href=\"https://www.hsph.harvard.edu/c-change/\" target=\"_blank\" rel=\"noopener\">Center for Climate, Health and the Global Environment\u003c/a> at Harvard University. And as the climate changes, temperatures spike and \u003ca href=\"https://science2017.globalchange.gov/chapter/6/\" target=\"_blank\" rel=\"noopener\">heat waves are more frequent\u003c/a>.\u003c/p>\n\u003cp>To learn more about how the heat influences young, healthy adults, Allen and his colleagues studied college students living in dorms during a summer heat wave in Boston.\u003c/p>\n\u003cp>Half of the students lived in buildings with central AC, where the indoor air temperature averaged 71 degrees. The other half lived in dorms with no AC, where air temperatures averaged almost 80 degrees.\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>\"In the morning, when they woke up, we pushed tests out to their cellphones,\" explains Allen. The students took two tests a day for 12 consecutive days.\u003c/p>\n\u003cp>One test, which included basic addition and subtraction, measured cognitive speed and memory. A second test assessed attention and processing speed.\u003c/p>\n\u003cp>\"We found that the students who were in the non-air-conditioned buildings actually had slower reaction times: 13 percent lower performance on basic arithmetic tests, and nearly a 10 percent reduction in the number of correct responses per minute,\" Allen explains.\u003c/p>\n\u003cp>The results, \u003ca href=\"http://journals.plos.org/plosmedicine/article?id=10.1371/journal.pmed.1002605\" target=\"_blank\" rel=\"noopener\">published in PLOS Medicine\u003c/a>, may come as a surprise. \"I think it's a little bit akin to the frog in the boiling water,\" Allen says. There's a \"slow, steady — largely imperceptible — rise in temperature, and you don't realize it's having an impact on you.\"\u003c/p>\n\u003cp>The findings add to a growing body of evidence that documents the effect of heat on mental performance, both in schools and workplaces.\u003c/p>\n\u003cp>For instance, a 2006 study from researchers at the Lawrence Berkeley National Lab found that when office temperatures rise above the mid-70s, \u003ca href=\"https://indoor.lbl.gov/publications/effect-temperature-task-performance\" target=\"_blank\" rel=\"noopener\">workers' performance begins to drop off\u003c/a>. Researchers reviewed multiple studies that evaluated performance on common office tasks\u003cem>.\u003c/em> The study found that worker productivity is highest at about 72 degrees. When temperatures exceeded the mid-80s, worker productivity decreased by about 9 percent.\u003c/p>\n\u003cp>Another, \u003ca href=\"https://www.sciencedirect.com/science/article/pii/S0360132316304723\" target=\"_blank\" rel=\"noopener\">more recent study\u003c/a> compared worker performance in green-certified buildings and typical office buildings. They found a dip in cognitive function linked to conditions in the indoor environment, including higher indoor temperatures and poor lighting.\u003c/p>\n\u003cp>And, when it comes to performance in the classroom, a study funded by the Harvard Environmental Economics Program finds that taking a standardized test on a very hot day is\u003ca href=\"https://scholar.harvard.edu/files/jisungpark/files/paper_nyc_aejep.pdf\" target=\"_blank\" rel=\"noopener\"> linked to poorer performance\u003c/a>. The study includes an analysis of test scores from students in New York City who take a series of high-school exams called the Regents Exams.\u003c/p>\n\u003cp>The author, R. Jisung Park, assistant professor at the University of California, Los Angeles, writes that compared with a 72-degree day, \"taking an exam on a 90◦F day leads to a 10.9 percent lower likelihood of passing a particular subject (e.g. Algebra), which in turn affects probability of graduation.\"\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>There's still a lot to learn about how our brains and bodies respond to heat. \"We all tend to think we can compensate, we can do just fine\" during heat waves says Allen. But he says the \"evidence shows that the indoor temperature can have a dramatic impact on our ability to be productive and learn.\"\u003c/p>\n\u003cdiv class=\"fullattribution\">Copyright 2018 NPR. To see more, visit http://www.npr.org/.\u003cimg src=\"https://www.google-analytics.com/__utm.gif?utmac=UA-5828686-4&utmdt=Heat+Making+You+Lethargic%3F+Research+Shows+It+Can+Slow+Your+Brain%2C+Too&utme=8(APIKey)9(MDAxOTAwOTE4MDEyMTkxMDAzNjczZDljZA004)\">\u003c/div>\n\n\u003c/div>\u003c/p>",
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"content": "\u003cp>Vicki Bartholomew started a support group for wives who are caring for a husband with Alzheimer's disease because she needed that sort of group herself.\u003c/p>\n\u003cp>They meet every month in a conference room at a new memory care facility in Nashville, Tenn., called \u003ca href=\"http://nashvillepublicradio.org/post/nashville-dementia-ward-aims-become-hub-memory-care-research#stream/0\" target=\"_blank\" rel=\"noopener\">Abe's Garden\u003c/a>, where Bartholomew's husband was one of the first residents — a Vietnam veteran and prominent attorney in Nashville.\u003c/p>\n\u003cp>\"My husband's still living, and now I'm in an even more difficult situation — I'm married, but I'm a widow,\" she says.\u003c/p>\n\u003cp>These women draw the shades and open up to each other in ways they can't with their lifelong friends.\u003c/p>\n\u003cp>\"They're still wonderful friends, but they didn't know how to handle this. It was hard for them, and as you all know, your friends don't come around as much as they used to,\" Bartholomew says. \"I was in bad shape. I didn't think I was — I did have health problems, and [now] I know I was depressed.\"\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>As the numbers of Americans afflicted with Alzheimer's disease continue to swell to an estimated 5.7 million, so do the legions of loved ones caring for friends and family members. The toll on Bartholomew's own mental health is one of the reasons the Alzheimer's Foundation of America focuses on the nation's estimated \u003ca href=\"https://www.alz.org/facts/\" target=\"_blank\" rel=\"noopener\">16 million unpaid caregivers\u003c/a>.\u003c/p>\n\u003cp>With no cure on the horizon, the foundation has been highlighting the necessity of better support for those caregivers through a \u003ca href=\"https://alzfdn.org/afa-educating-america-tour/\">national tour\u003c/a>. It stopped in Nashville earlier this spring, was in Tempe, Ariz., in June and heads to Fairfax, Va., in September; the tour includes at least six more cities in the fall.\u003c/p>\n\u003cp>At the live events, Alzheimer's researchers and clinicians offer guidance on a number of topics, including how to ensure safety for patients at home, care planning and even how to \u003ca href=\"https://alzfdn.org/event/afa-educating-america-tour-tempe/\" target=\"_blank\" rel=\"noopener\">entertain someone\u003c/a> with memory loss.\u003c/p>\n\u003cp>The organization promotes in-person and \u003ca href=\"https://alzfdn.org/alzheimers-foundation-americas-national-toll-free-helpline-expanding-seven-days-week/\" target=\"_blank\" rel=\"noopener\">telephone support groups\u003c/a>, since being a caregiver is often a barrier to getting out of the house alone. But even virtual support systems have shown some effectiveness at reducing loneliness, stress and depression \u003ca href=\"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4668714/\" target=\"_blank\" rel=\"noopener\">in a small 2014 study.\u003c/a>\u003c/p>\n\u003cp>\"We have to do everything we can to educate a caregiver, to provide them with the best practices on caring for somebody,\" says \u003ca href=\"https://alzfdn.org/team_member/charles-j-fuschillo-jr/\" target=\"_blank\" rel=\"noopener\">Charles J. Fuschillo Jr.,\u003c/a> the Alzheimer's Foundation of America's CEO.\u003c/p>\n\u003cp>For example, the AFA \u003ca href=\"https://alzfdn.org/caregiving-resources/fact-sheets-information/\" target=\"_blank\" rel=\"noopener\">recommends\u003c/a> that family members:\u003c/p>\n\u003cul>\n\u003cli>Feed Alzheimer's patients one food at a time. \"A busy plate can be confusing,\" the group says.\u003c/li>\n\u003cli>Mark rooms in the house with signs to avoid unnecessary confusion.\u003c/li>\n\u003cli>Remind a person with Alzheimer's to use the toilet; don't wait for them to ask.\u003c/li>\n\u003cli>When traveling, stick with familiar destinations.\u003c/li>\n\u003cli>Watch for a cough while eating; it can signal a swallowing disorder in people with dementia.\u003c/li>\n\u003cli>Schedule overnight stays at a memory-care facility so the caregiver gets some respite.\u003c/li>\n\u003c/ul>\n\u003cp>Just as important, Fuschillo says, \"We want to do everything we can to avoid caregiver burnout.\"\u003c/p>\n\u003cp>The breaking point sneaks up on even the most committed caregiver, say Alzheimer's advocates, especially as the nights grow more sleepless. Alzheimer's patients can tend to pace, or wake up their partner every few minutes. They can even become violent. Or, perhaps worse, they can leave the house.\u003c/p>\n\u003cp>\"And I've had some issues at night that I had to take care of alone,\" says Pam Hawkins, whose husband has Alzheimer's. \"But I'm not ready to have anyone there at night.\"\u003c/p>\n\u003cp>For now, she says, her husband usually sleeps all night. And if there's a problem, her son-in-law is 15 minutes away.\u003c/p>\n\u003cp>She's had to hire caregivers during the day. Knowing how to find and hire the right person is a shared concern by Alzheimer's family members that has inspired an entire\u003ca href=\"https://www.alz.org/national/documents/topicsheet_homehealth.pdf\" target=\"_blank\" rel=\"noopener\"> checklist\u003c/a> for navigating the process. The tips include these: Interview the aide in the home. Over-share information about the patient. Ask what kind of quality control a supervisor will provide.\u003c/p>\n\u003cp>Hawkins is adamant about keeping her husband at home, whatever the cost.\u003c/p>\n\u003cp>\"He's not going anywhere,\" she says. \"He's staying at our home until he moves to heaven. We made that decision a long time ago.\"\u003c/p>\n\u003cp>But many caregivers have no choice.\u003c/p>\n\u003cp>April Simpkins says tending to her husband became all consuming, and she's young enough that she still needs to keep her job; she works at a local university.\u003c/p>\n\u003cp>\"It was not possible for us to keep Joe at home,\" she says.\u003c/p>\n\u003cp>Simpkins found she'd often have to call her husband's siblings to settle him down over the phone. One night, she had to dial 911 when he kept yelling in the hallways of their condo building.\u003c/p>\n\u003cp>And yet she felt some societal pressure that she wasn't doing enough.\u003c/p>\n\u003cp>\"There's a lot of ... glory given to the whole idea of someone being long-suffering and staying at home and giving up their life, basically, to care for their loved one,\" Simpkins says. \"It makes it harder for people who can't do that.\"\u003c/p>\n\u003cp>Everyone around the table nods in agreement. Whatever stage of illness their loved one is experiencing, these caregivers understand the complicated existence that many have dubbed \"the long goodbye.\"\u003c/p>\n\u003cp>Along with sharing the sorrow, they find a way to share in the humor of it all — one woman says her husband wears a laundry-basket's-worth of shirts and pants because he forgets he's already gotten dressed. Even tips on how to reduce the odor from incontinence are offered with a loving laugh.\u003c/p>\n\u003cp>The support group ends with hugs. Some women head for the parking lot. Others buzz through the locked doors to see their husbands.\u003c/p>\n\u003cp>Simpkins sits down for lunch with Joe, who is a former state employee and a youthful-looking 66 years old. She drapes an arm around his slumping shoulders and assists him as he spears a cold strawberry with his fork.\u003c/p>\n\u003cp>\"You know, there are some days,\" she says, interrupted by a random reflection from Joe. \"Yeah, some days are clearer than others.\"\u003c/p>\n\u003cp>Simpkins tries to stop by to see her husband every day. But it's a wicked kind of blessing, she says, that when she misses a visit, Joe no longer notices.\u003c/p>\n\u003cp>[ad floatright]\u003c/p>\n\u003cp>\u003cem>This story is part of NPR's reporting partnership with Nashville Public Radio and Kaiser Health News.\u003c/em>\u003c/p>\n\u003cdiv class=\"fullattribution\">Copyright 2018 Nashville Public Radio. To see more, visit \u003ca href=\"http://www.wpln.org/\" target=\"_blank\" rel=\"noopener\">Nashville Public Radio\u003c/a>.\u003cimg src=\"https://www.google-analytics.com/__utm.gif?utmac=UA-5828686-4&utmdt=Family+Caregivers+Exchange+Tips%2C+Share+Stories+To+Ease+Alzheimer%27s+Losses&utme=8(APIKey)9(MDAxOTAwOTE4MDEyMTkxMDAzNjczZDljZA004)\">\u003c/div>\n\n",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp>Vicki Bartholomew started a support group for wives who are caring for a husband with Alzheimer's disease because she needed that sort of group herself.\u003c/p>\n\u003cp>They meet every month in a conference room at a new memory care facility in Nashville, Tenn., called \u003ca href=\"http://nashvillepublicradio.org/post/nashville-dementia-ward-aims-become-hub-memory-care-research#stream/0\" target=\"_blank\" rel=\"noopener\">Abe's Garden\u003c/a>, where Bartholomew's husband was one of the first residents — a Vietnam veteran and prominent attorney in Nashville.\u003c/p>\n\u003cp>\"My husband's still living, and now I'm in an even more difficult situation — I'm married, but I'm a widow,\" she says.\u003c/p>\n\u003cp>These women draw the shades and open up to each other in ways they can't with their lifelong friends.\u003c/p>\n\u003cp>\"They're still wonderful friends, but they didn't know how to handle this. It was hard for them, and as you all know, your friends don't come around as much as they used to,\" Bartholomew says. \"I was in bad shape. I didn't think I was — I did have health problems, and [now] I know I was depressed.\"\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>As the numbers of Americans afflicted with Alzheimer's disease continue to swell to an estimated 5.7 million, so do the legions of loved ones caring for friends and family members. The toll on Bartholomew's own mental health is one of the reasons the Alzheimer's Foundation of America focuses on the nation's estimated \u003ca href=\"https://www.alz.org/facts/\" target=\"_blank\" rel=\"noopener\">16 million unpaid caregivers\u003c/a>.\u003c/p>\n\u003cp>With no cure on the horizon, the foundation has been highlighting the necessity of better support for those caregivers through a \u003ca href=\"https://alzfdn.org/afa-educating-america-tour/\">national tour\u003c/a>. It stopped in Nashville earlier this spring, was in Tempe, Ariz., in June and heads to Fairfax, Va., in September; the tour includes at least six more cities in the fall.\u003c/p>\n\u003cp>At the live events, Alzheimer's researchers and clinicians offer guidance on a number of topics, including how to ensure safety for patients at home, care planning and even how to \u003ca href=\"https://alzfdn.org/event/afa-educating-america-tour-tempe/\" target=\"_blank\" rel=\"noopener\">entertain someone\u003c/a> with memory loss.\u003c/p>\n\u003cp>The organization promotes in-person and \u003ca href=\"https://alzfdn.org/alzheimers-foundation-americas-national-toll-free-helpline-expanding-seven-days-week/\" target=\"_blank\" rel=\"noopener\">telephone support groups\u003c/a>, since being a caregiver is often a barrier to getting out of the house alone. But even virtual support systems have shown some effectiveness at reducing loneliness, stress and depression \u003ca href=\"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4668714/\" target=\"_blank\" rel=\"noopener\">in a small 2014 study.\u003c/a>\u003c/p>\n\u003cp>\"We have to do everything we can to educate a caregiver, to provide them with the best practices on caring for somebody,\" says \u003ca href=\"https://alzfdn.org/team_member/charles-j-fuschillo-jr/\" target=\"_blank\" rel=\"noopener\">Charles J. Fuschillo Jr.,\u003c/a> the Alzheimer's Foundation of America's CEO.\u003c/p>\n\u003cp>For example, the AFA \u003ca href=\"https://alzfdn.org/caregiving-resources/fact-sheets-information/\" target=\"_blank\" rel=\"noopener\">recommends\u003c/a> that family members:\u003c/p>\n\u003cul>\n\u003cli>Feed Alzheimer's patients one food at a time. \"A busy plate can be confusing,\" the group says.\u003c/li>\n\u003cli>Mark rooms in the house with signs to avoid unnecessary confusion.\u003c/li>\n\u003cli>Remind a person with Alzheimer's to use the toilet; don't wait for them to ask.\u003c/li>\n\u003cli>When traveling, stick with familiar destinations.\u003c/li>\n\u003cli>Watch for a cough while eating; it can signal a swallowing disorder in people with dementia.\u003c/li>\n\u003cli>Schedule overnight stays at a memory-care facility so the caregiver gets some respite.\u003c/li>\n\u003c/ul>\n\u003cp>Just as important, Fuschillo says, \"We want to do everything we can to avoid caregiver burnout.\"\u003c/p>\n\u003cp>The breaking point sneaks up on even the most committed caregiver, say Alzheimer's advocates, especially as the nights grow more sleepless. Alzheimer's patients can tend to pace, or wake up their partner every few minutes. They can even become violent. Or, perhaps worse, they can leave the house.\u003c/p>\n\u003cp>\"And I've had some issues at night that I had to take care of alone,\" says Pam Hawkins, whose husband has Alzheimer's. \"But I'm not ready to have anyone there at night.\"\u003c/p>\n\u003cp>For now, she says, her husband usually sleeps all night. And if there's a problem, her son-in-law is 15 minutes away.\u003c/p>\n\u003cp>She's had to hire caregivers during the day. Knowing how to find and hire the right person is a shared concern by Alzheimer's family members that has inspired an entire\u003ca href=\"https://www.alz.org/national/documents/topicsheet_homehealth.pdf\" target=\"_blank\" rel=\"noopener\"> checklist\u003c/a> for navigating the process. The tips include these: Interview the aide in the home. Over-share information about the patient. Ask what kind of quality control a supervisor will provide.\u003c/p>\n\u003cp>Hawkins is adamant about keeping her husband at home, whatever the cost.\u003c/p>\n\u003cp>\"He's not going anywhere,\" she says. \"He's staying at our home until he moves to heaven. We made that decision a long time ago.\"\u003c/p>\n\u003cp>But many caregivers have no choice.\u003c/p>\n\u003cp>April Simpkins says tending to her husband became all consuming, and she's young enough that she still needs to keep her job; she works at a local university.\u003c/p>\n\u003cp>\"It was not possible for us to keep Joe at home,\" she says.\u003c/p>\n\u003cp>Simpkins found she'd often have to call her husband's siblings to settle him down over the phone. One night, she had to dial 911 when he kept yelling in the hallways of their condo building.\u003c/p>\n\u003cp>And yet she felt some societal pressure that she wasn't doing enough.\u003c/p>\n\u003cp>\"There's a lot of ... glory given to the whole idea of someone being long-suffering and staying at home and giving up their life, basically, to care for their loved one,\" Simpkins says. \"It makes it harder for people who can't do that.\"\u003c/p>\n\u003cp>Everyone around the table nods in agreement. Whatever stage of illness their loved one is experiencing, these caregivers understand the complicated existence that many have dubbed \"the long goodbye.\"\u003c/p>\n\u003cp>Along with sharing the sorrow, they find a way to share in the humor of it all — one woman says her husband wears a laundry-basket's-worth of shirts and pants because he forgets he's already gotten dressed. Even tips on how to reduce the odor from incontinence are offered with a loving laugh.\u003c/p>\n\u003cp>The support group ends with hugs. Some women head for the parking lot. Others buzz through the locked doors to see their husbands.\u003c/p>\n\u003cp>Simpkins sits down for lunch with Joe, who is a former state employee and a youthful-looking 66 years old. She drapes an arm around his slumping shoulders and assists him as he spears a cold strawberry with his fork.\u003c/p>\n\u003cp>\"You know, there are some days,\" she says, interrupted by a random reflection from Joe. \"Yeah, some days are clearer than others.\"\u003c/p>\n\u003cp>Simpkins tries to stop by to see her husband every day. But it's a wicked kind of blessing, she says, that when she misses a visit, Joe no longer notices.\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>\u003cem>This story is part of NPR's reporting partnership with Nashville Public Radio and Kaiser Health News.\u003c/em>\u003c/p>\n\u003cdiv class=\"fullattribution\">Copyright 2018 Nashville Public Radio. To see more, visit \u003ca href=\"http://www.wpln.org/\" target=\"_blank\" rel=\"noopener\">Nashville Public Radio\u003c/a>.\u003cimg src=\"https://www.google-analytics.com/__utm.gif?utmac=UA-5828686-4&utmdt=Family+Caregivers+Exchange+Tips%2C+Share+Stories+To+Ease+Alzheimer%27s+Losses&utme=8(APIKey)9(MDAxOTAwOTE4MDEyMTkxMDAzNjczZDljZA004)\">\u003c/div>\n\n\u003c/div>\u003c/p>",
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"disqusTitle": "Mother: Caring for Girl Amid Brain-Death Debate ‘Worth It’",
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"content": "\u003cp>The mother of a girl at the center of a medical and religious debate over brain death said she does not regret moving from California to New Jersey so her daughter could receive care after being declared dead.[contextly_sidebar id=\"23Ay0lmvAahQPeVvigzIETSKtEkm43UJ\"]\u003c/p>\n\u003cp>Nailah Winkfield told reporters Tuesday that she gave up everything for daughter Jahi McMath.\u003c/p>\n\u003cp>“Everything that I did, from selling my house, to quitting my job, to moving across the country and taking all that time away from my family, it was all worth it,” Winkfield said.\u003c/p>\n\u003cp>She has said doctors declared Jahi dead on June 22 from excessive bleeding and liver failure after an operation to treat an intestinal issue.\u003c/p>\n\u003cp>Jahi had been declared dead in December 2013 at age 13 after suffering irreversible brain damage during surgery in California to remove her tonsils. A coroner signed a death certificate.\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>Winkfield refused to accept the conclusion and took Jahi to New Jersey, which accommodates religions that don’t recognize brain death.\u003c/p>\n\u003cp>Winkfield said Tuesday that her daughter grew and went through puberty — evidence she was not dead.\u003c/p>\n\u003cp>“There’s no way in the world that I would be holding onto a corpse for 4½ years,” she said.\u003c/p>\n\u003cp>She also described her final moments with Jahi. Winkfield said she gave her daughter permission to “go” if she was tired, telling her not to worry about her mom.[contextly_sidebar id=\"aYsDGYXXZXPDLEprrXkMdRXaPOQCyavj\"]\u003c/p>\n\u003cp>“I said, ‘You have my permission. You can go,’” she said. “I said, ‘My husband will see about me, your siblings will see about me. Don’t worry.’”\u003c/p>\n\u003cp>She said Jahi died hours later.\u003c/p>\n\u003cp>“It’s going to be hard without her,” she said. “She was a sweet girl.”\u003c/p>\n\u003cp>Jahi will be buried Friday in Hayward, California, the San Francisco Chronicle \u003ca href=\"https://www.sfgate.com/bayarea/article/Family-of-Jahi-McMath-gets-approval-to-bury-their-13047928.php\">reported\u003c/a> .\u003c/p>\n\u003cp>Conservative religious groups rallied behind Winkfield and helped raise money for Jahi’s continued care.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>Winkfield and her lawyers have been trying to rescind the California death certificate as part of a medical malpractice lawsuit against the hospital where Jahi had her tonsillectomy.\u003c/p>\n\n",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp>The mother of a girl at the center of a medical and religious debate over brain death said she does not regret moving from California to New Jersey so her daughter could receive care after being declared dead.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>Nailah Winkfield told reporters Tuesday that she gave up everything for daughter Jahi McMath.\u003c/p>\n\u003cp>“Everything that I did, from selling my house, to quitting my job, to moving across the country and taking all that time away from my family, it was all worth it,” Winkfield said.\u003c/p>\n\u003cp>She has said doctors declared Jahi dead on June 22 from excessive bleeding and liver failure after an operation to treat an intestinal issue.\u003c/p>\n\u003cp>Jahi had been declared dead in December 2013 at age 13 after suffering irreversible brain damage during surgery in California to remove her tonsils. A coroner signed a death certificate.\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>Winkfield refused to accept the conclusion and took Jahi to New Jersey, which accommodates religions that don’t recognize brain death.\u003c/p>\n\u003cp>Winkfield said Tuesday that her daughter grew and went through puberty — evidence she was not dead.\u003c/p>\n\u003cp>“There’s no way in the world that I would be holding onto a corpse for 4½ years,” she said.\u003c/p>\n\u003cp>She also described her final moments with Jahi. Winkfield said she gave her daughter permission to “go” if she was tired, telling her not to worry about her mom.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>“I said, ‘You have my permission. You can go,’” she said. “I said, ‘My husband will see about me, your siblings will see about me. Don’t worry.’”\u003c/p>\n\u003cp>She said Jahi died hours later.\u003c/p>\n\u003cp>“It’s going to be hard without her,” she said. “She was a sweet girl.”\u003c/p>\n\u003cp>Jahi will be buried Friday in Hayward, California, the San Francisco Chronicle \u003ca href=\"https://www.sfgate.com/bayarea/article/Family-of-Jahi-McMath-gets-approval-to-bury-their-13047928.php\">reported\u003c/a> .\u003c/p>\n\u003cp>Conservative religious groups rallied behind Winkfield and helped raise money for Jahi’s continued care.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>Winkfield and her lawyers have been trying to rescind the California death certificate as part of a medical malpractice lawsuit against the hospital where Jahi had her tonsillectomy.\u003c/p>\n\n\u003c/div>\u003c/p>",
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"disqusTitle": "Can Zapping People’s Brains Reduce Violence? Controversial Study Sees Potential",
"title": "Can Zapping People’s Brains Reduce Violence? Controversial Study Sees Potential",
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"content": "\u003cp>The study participants read short accounts of violent behavior: In one, a man smashed a beer bottle over someone’s head; in another, an assailant raped an acquaintance.[contextly_sidebar id=\"gKdycmp0KoVJxcxC1F1K6WdKxrJ9rXZI\"]\u003c/p>\n\u003cp>They were then asked: \u003cem>Would you do that?\u003c/em>\u003c/p>\n\u003cp>The day before, half of them had had the frontmost region of their brains, responsible for such high-level functions as impulse control and moral judgments, electrically stimulated; the other half had not.\u003c/p>\n\u003cp>The people whose prefrontal cortex was stimulated reported roughly half the likelihood of committing a violent act like the ones they watched, scientists at the University of Pennsylvania reported on Monday; they said they found such physical and sexual violence more morally wrong, compared with the control group.\u003c/p>\n\u003cp>The researchers hailed their results as evidence that “increasing activity in the prefrontal cortex can reduce intentions to commit aggression and enhance perceptions of moral judgment,” as they wrote in the Journal of Neuroscience.\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>Other experts said the results were far less than advertised.\u003c/p>\n\u003cp>“We’re a long way from ‘Clockwork Orange,’” said Dr. Paul Appelbaum, director of the Division of Law, Ethics, and Psychiatry at Columbia University’s Vagelos College of Physicians and Surgeons.\u003c/p>\n\u003cp>For the study, the Penn researchers randomly assigned 81 healthy adults (the average ago was 20) to receive “transcranial direct-current stimulation” of the dorsolateral prefrontal cortex, behind the top of the forehead, for 20 minutes via two scalp electrodes, or to get a low current for 30 seconds and then nothing, also via electrodes. Participants couldn’t tell if they were receiving the stimulation or not.\u003c/p>\n\u003cp>The researchers aimed at the dorsolateral prefrontal cortex because dozens of small neuro-imaging studies have \u003ca href=\"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2784035/\" target=\"_blank\" rel=\"noopener\">reported\u003c/a> that in people deemed antisocial or aggressive or both, this region is typically smaller and less active than in other people. Such studies can’t tell whether an impaired prefrontal cortex causes aggression or vice versa, so the Penn study, a registered \u003ca href=\"https://clinicaltrials.gov/ct2/show/NCT02427672\" target=\"_blank\" rel=\"noopener\">clinical trial\u003c/a>, tried to figure that out.[contextly_sidebar id=\"dnHIsBbKjxIupKuLmg7VYu4mKCZSmb4l\"]\u003c/p>\n\u003cp>The next day, participants read one vignette about the beer-bottle assault and one about the rape, and rated how likely they were (from “no chance” to “definitely”) to act as the assaulter did. The brain-stimulated group reported a 47 percent lower likelihood that they would commit the non-sexual physical assault (1.15 vs. 2.19 on a scale of 0 to 10) and a 70 percent lower likelihood of committing the sexual assault (0.26 vs. 0.86) compared with the control group. The brain-stimulated group also rated the assaults as more morally wrong than the control group did.\u003c/p>\n\u003cp>But while the brain stimulation decreased people’s self-reported likelihood of committing assault, their actions indicated otherwise. In the only assessment of actual behavior, the participants got to stick pins into a computer image of a doll representing a close friend, a common lab test of violent tendencies. Those whose prefrontal cortex was stimulated stuck in slightly more pins than the control group.\u003c/p>\n\u003cp>“What people say they will do with regard to violence and what they actually do may be two different things,” said Appelbaum. “Whether actual violence would be reduced [by brain stimulation] is unknown,” but “the data suggest no impact” on actual aggressive behavior.\u003c/p>\n\u003cp>Olivia Choy, the paper’s lead author and now a psychologist at Nanyang Technological University in Singapore, emphasized that the participants got only one 20-minute stimulation session. “It might be that repeated sessions over a longer time period could produce changes in behavior, but changes in behavior start with changing intent,” she said.\u003c/p>\n\u003cp>“We’re trying to find benign biological interventions [for criminal violence] that society will accept,” Penn psychologist Adrian Raine, who has studied the brain basis of violence and psychopathy for decades, said in a statement. “Transcranial direct-current stimulation is minimal risk. This isn’t a frontal lobotomy.”\u003c/p>\n\u003cp>Robin Mackenzie, a scholar of medical law and ethics at England’s University of Kent, called the study “promising and suggestive,” saying it “advances the field of biological interventions on antisocial and aggressive behavior.”\u003c/p>\n\u003cp>But she, too, noted several concerns. One is that the brain-stimulated group had 24 women and 15 men, compared with 21 and 21 in the control group. That raises the question of whether the zapped group’s lower likelihood of endorsing rape and beer-bottle attacks might be driven by gender differences, especially on what constitutes rape, Mackenzie said: “Asking female participants to identify with a rapist to assess how likely they would be to rape a woman is inherently different from asking male participants to do so….It is at the very least possible that the gender distribution could have skewed the results.”[contextly_sidebar id=\"APQk5r3dfTy6mLZEMSYpZsZfNmt4Z4Oq\"]\u003c/p>\n\u003cp>In addition, the study participants had no history of psychiatric or neurologic disease or injury, all of which are associated with violent tendencies. But the brain mechanisms causing that violence in these groups may differ from the mechanisms in normal people. Results on healthy, non-violent college students therefore might not apply to people with such brain abnormalities. “How far tDCS could affect [such people] is an open question,” Mackenzie said.\u003c/p>\n\u003cp>And even if stimulation of the prefrontal cortex makes people express a heightened sense that violence is wrong, when they are asked their views the next day, it remains to be seen whether any such effect would last, or if people would have to be zapped frequently.\u003c/p>\n\u003cp>Regardless of its shortcomings, the study worries some experts. If governments adopt brain zapping for violent offenders, would it be voluntary or, just as some states and countries mandate chemical castration for some male sex offenders, mandatory? And would governments be tempted to extend its use beyond violence, such as “to induce passivity in politically unruly groups?,” Appelbaum asked.\u003c/p>\n\u003cp>“Neurotechnologies which affect identity, ways of being, and thought processes are particularly tempting for authorities seeking to secure control and cut costs,” Mackenzie said.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>\u003cem>This\u003ca href=\"https://www.statnews.com/2018/07/02/brain-electric-stimulation-violence/\" target=\"_blank\" rel=\"noopener\"> story\u003c/a> was originally published by STAT, an online publication of Boston Globe Media that covers health, medicine, and scientific discovery.\u003c/em>\u003c/p>\n\n",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp>The study participants read short accounts of violent behavior: In one, a man smashed a beer bottle over someone’s head; in another, an assailant raped an acquaintance.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>They were then asked: \u003cem>Would you do that?\u003c/em>\u003c/p>\n\u003cp>The day before, half of them had had the frontmost region of their brains, responsible for such high-level functions as impulse control and moral judgments, electrically stimulated; the other half had not.\u003c/p>\n\u003cp>The people whose prefrontal cortex was stimulated reported roughly half the likelihood of committing a violent act like the ones they watched, scientists at the University of Pennsylvania reported on Monday; they said they found such physical and sexual violence more morally wrong, compared with the control group.\u003c/p>\n\u003cp>The researchers hailed their results as evidence that “increasing activity in the prefrontal cortex can reduce intentions to commit aggression and enhance perceptions of moral judgment,” as they wrote in the Journal of Neuroscience.\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>Other experts said the results were far less than advertised.\u003c/p>\n\u003cp>“We’re a long way from ‘Clockwork Orange,’” said Dr. Paul Appelbaum, director of the Division of Law, Ethics, and Psychiatry at Columbia University’s Vagelos College of Physicians and Surgeons.\u003c/p>\n\u003cp>For the study, the Penn researchers randomly assigned 81 healthy adults (the average ago was 20) to receive “transcranial direct-current stimulation” of the dorsolateral prefrontal cortex, behind the top of the forehead, for 20 minutes via two scalp electrodes, or to get a low current for 30 seconds and then nothing, also via electrodes. Participants couldn’t tell if they were receiving the stimulation or not.\u003c/p>\n\u003cp>The researchers aimed at the dorsolateral prefrontal cortex because dozens of small neuro-imaging studies have \u003ca href=\"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2784035/\" target=\"_blank\" rel=\"noopener\">reported\u003c/a> that in people deemed antisocial or aggressive or both, this region is typically smaller and less active than in other people. Such studies can’t tell whether an impaired prefrontal cortex causes aggression or vice versa, so the Penn study, a registered \u003ca href=\"https://clinicaltrials.gov/ct2/show/NCT02427672\" target=\"_blank\" rel=\"noopener\">clinical trial\u003c/a>, tried to figure that out.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>The next day, participants read one vignette about the beer-bottle assault and one about the rape, and rated how likely they were (from “no chance” to “definitely”) to act as the assaulter did. The brain-stimulated group reported a 47 percent lower likelihood that they would commit the non-sexual physical assault (1.15 vs. 2.19 on a scale of 0 to 10) and a 70 percent lower likelihood of committing the sexual assault (0.26 vs. 0.86) compared with the control group. The brain-stimulated group also rated the assaults as more morally wrong than the control group did.\u003c/p>\n\u003cp>But while the brain stimulation decreased people’s self-reported likelihood of committing assault, their actions indicated otherwise. In the only assessment of actual behavior, the participants got to stick pins into a computer image of a doll representing a close friend, a common lab test of violent tendencies. Those whose prefrontal cortex was stimulated stuck in slightly more pins than the control group.\u003c/p>\n\u003cp>“What people say they will do with regard to violence and what they actually do may be two different things,” said Appelbaum. “Whether actual violence would be reduced [by brain stimulation] is unknown,” but “the data suggest no impact” on actual aggressive behavior.\u003c/p>\n\u003cp>Olivia Choy, the paper’s lead author and now a psychologist at Nanyang Technological University in Singapore, emphasized that the participants got only one 20-minute stimulation session. “It might be that repeated sessions over a longer time period could produce changes in behavior, but changes in behavior start with changing intent,” she said.\u003c/p>\n\u003cp>“We’re trying to find benign biological interventions [for criminal violence] that society will accept,” Penn psychologist Adrian Raine, who has studied the brain basis of violence and psychopathy for decades, said in a statement. “Transcranial direct-current stimulation is minimal risk. This isn’t a frontal lobotomy.”\u003c/p>\n\u003cp>Robin Mackenzie, a scholar of medical law and ethics at England’s University of Kent, called the study “promising and suggestive,” saying it “advances the field of biological interventions on antisocial and aggressive behavior.”\u003c/p>\n\u003cp>But she, too, noted several concerns. One is that the brain-stimulated group had 24 women and 15 men, compared with 21 and 21 in the control group. That raises the question of whether the zapped group’s lower likelihood of endorsing rape and beer-bottle attacks might be driven by gender differences, especially on what constitutes rape, Mackenzie said: “Asking female participants to identify with a rapist to assess how likely they would be to rape a woman is inherently different from asking male participants to do so….It is at the very least possible that the gender distribution could have skewed the results.”\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>In addition, the study participants had no history of psychiatric or neurologic disease or injury, all of which are associated with violent tendencies. But the brain mechanisms causing that violence in these groups may differ from the mechanisms in normal people. Results on healthy, non-violent college students therefore might not apply to people with such brain abnormalities. “How far tDCS could affect [such people] is an open question,” Mackenzie said.\u003c/p>\n\u003cp>And even if stimulation of the prefrontal cortex makes people express a heightened sense that violence is wrong, when they are asked their views the next day, it remains to be seen whether any such effect would last, or if people would have to be zapped frequently.\u003c/p>\n\u003cp>Regardless of its shortcomings, the study worries some experts. If governments adopt brain zapping for violent offenders, would it be voluntary or, just as some states and countries mandate chemical castration for some male sex offenders, mandatory? And would governments be tempted to extend its use beyond violence, such as “to induce passivity in politically unruly groups?,” Appelbaum asked.\u003c/p>\n\u003cp>“Neurotechnologies which affect identity, ways of being, and thought processes are particularly tempting for authorities seeking to secure control and cut costs,” Mackenzie said.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>\u003cem>This\u003ca href=\"https://www.statnews.com/2018/07/02/brain-electric-stimulation-violence/\" target=\"_blank\" rel=\"noopener\"> story\u003c/a> was originally published by STAT, an online publication of Boston Globe Media that covers health, medicine, and scientific discovery.\u003c/em>\u003c/p>\n\n\u003c/div>\u003c/p>",
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"content": "\u003cp>Doctors have long known that separating families and other traumatic events can damage children’s well-being. More recent research has shed some light on how that may happen: Severe early adversity may cause brain changes and “toxic stress,” resulting in lasting psychological and physical health problems.\u003c/p>\n\u003cp>A “zero-tolerance” immigration policy announced this spring by the Trump Administration has separated 2,300 children from the adults they were traveling with and sent them to shelters and foster homes across the country, according to the government. The administration has 30 days to reunite families, but it remains unclear whether it can meet that deadline.\u003c/p>\n\u003cp>Here’s what evidence shows this kind of separation can do and how some experts think immigrant children may be at risk.\u003c/p>\n\u003cp>\u003cstrong>The Stress Response\u003c/strong>\u003c/p>\n\u003cp>Stress is a normal response to challenging or threatening circumstances that cause the brain to trigger the body’s “fight or flight” mode. Stress hormones and chemicals are released that increase heart rate, blood pressure, alertness and energy levels.\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>Levels typically return to normal when the threat passes.\u003c/p>\n\u003cp>When the threat is ongoing — a result of things like war, famine, poverty, natural disasters, family discord or neighborhood violence — the stress system may remain on high alert, sometimes triggering anxiety, behavior problems, stomach aches, sleep problems and other mental and physical symptoms.\u003c/p>\n\u003cp>Experts believe that contact with a loving, nurturing parent or caregiver can help children weather stress and reduce chances for lasting ill effects. A recent U.S. study of toddlers from families living in poverty is an example. Youngsters who had a strong parent relationship had lower stress-hormone levels when they arrived at a clinic to get vaccinations than those without that parent buffer.\u003c/p>\n\u003cp>\u003cstrong>What's Toxic Stress? \u003c/strong>\u003c/p>\n\u003cp>Scientists increasingly believe that persistent stress, particularly when there’s no nurturing parent to help kids cope, can cause stress hormones and internal inflammatory markers to smolder at elevated levels, raising risks for later heart disease, diabetes and other health problems.\u003c/p>\n\u003cp>The science is not settled, but some studies also have shown that persistent stress may alter brain structure in regions affecting emotions and regulating behavior. Imaging studies have found these regions are smaller than usual in severely traumatized children.\u003c/p>\n\u003cp>Harvard University neuroscientist Charles Nelson says children younger than 3, with rapidly developing brains, are most at risk for the toxic effects of ongoing stress. In his imaging studies, school-aged Romanian orphans sent to live with foster families had brains with less gray matter than orphans sent to nurturing foster homes before age 2.\u003c/p>\n\u003cp>\u003cstrong>What About Migrant Children \u003c/strong>\u003c/p>\n\u003cp>Central American children arriving with their families at the southern U.S. border have already endured the trauma of leaving their homes, some after violence or other threats, and faced the additional trauma of an arduous journey north, Nelson said Wednesday. “That may increase their susceptibility to the hazards of separation at the border,” he said.\u003c/p>\n\u003cp>Nelson said the images he’s seen of U.S. detention centers housing children remind him of his research with Romanian children. In a recent research newsletter, Nelson wrote, “The lessons we learned then taught us that housing children in institution-like settings, with rotating shifts of caregivers and unfavorable ratios of caregivers to children, may cause severe and permanent damage to their minds and bodies.”\u003c/p>\n\u003cp>“This is what science has taught us about separation,” he said Wednesday.\u003c/p>\n\u003cp>\u003cstrong>What Doctors Say \u003c/strong>\u003c/p>\n\u003cp>In 2017, a year before the separation policy was announced, the American Academy of Pediatrics advised against holding migrant children in detention centers and said they should receive health care and treatment to “support their well-being.”\u003c/p>\n\u003cp>The \u003ca href=\"https://www.aap.org/en-us/about-the-aap/aap-press-room/Pages/AAPStatementOpposingBorderSecurityandImmigrationReformAct.aspx\">academy\u003c/a> and several mental health \u003ca href=\"http://www.apa.org/news/press/releases/2018/06/family-separation-policy.aspx\">groups\u003c/a> recently denounced the separation policy and cautiously supported last week’s executive order to overturn it. Tuesday’s order from a federal judge mandating that thousands of families be reunited within 30 days is an important step, the academy said in a joint statement with UNICEF USA, but it won’t undo the trauma separated children have already faced, said Academy President Dr. Colleen Kraft.\u003c/p>\n\u003cp>Even if they are eventually reunited, these families will need “trauma-informed” care to help them cope, Kraft said.\u003c/p>\n\u003cp>In their statement Wednesday, the academy and UNICEF USA said their organizations “stand ready to assist in reunifying families and supporting their needs. Pediatricians across the country are prepared to provide care for immigrant children as they enter our communities.”\u003c/p>\n\u003cp>Kraft attempted to enter a children’s shelter in McAllen, Texas on Wednesday to see what attempts were being made to reunite children and parents but said authorities turned her away. During an April visit to the same shelter, Kraft saw children who were “eerily quiet” and one who was crying inconsolably while attendants watched.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>“Staff in the room were not allowed to pick her up and hold her,” Kraft said. It’s that kind of absence of nurturing contact that experts think worsens the effects of traumatic stress.\u003c/p>\n\n",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp>Doctors have long known that separating families and other traumatic events can damage children’s well-being. More recent research has shed some light on how that may happen: Severe early adversity may cause brain changes and “toxic stress,” resulting in lasting psychological and physical health problems.\u003c/p>\n\u003cp>A “zero-tolerance” immigration policy announced this spring by the Trump Administration has separated 2,300 children from the adults they were traveling with and sent them to shelters and foster homes across the country, according to the government. The administration has 30 days to reunite families, but it remains unclear whether it can meet that deadline.\u003c/p>\n\u003cp>Here’s what evidence shows this kind of separation can do and how some experts think immigrant children may be at risk.\u003c/p>\n\u003cp>\u003cstrong>The Stress Response\u003c/strong>\u003c/p>\n\u003cp>Stress is a normal response to challenging or threatening circumstances that cause the brain to trigger the body’s “fight or flight” mode. Stress hormones and chemicals are released that increase heart rate, blood pressure, alertness and energy levels.\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>Levels typically return to normal when the threat passes.\u003c/p>\n\u003cp>When the threat is ongoing — a result of things like war, famine, poverty, natural disasters, family discord or neighborhood violence — the stress system may remain on high alert, sometimes triggering anxiety, behavior problems, stomach aches, sleep problems and other mental and physical symptoms.\u003c/p>\n\u003cp>Experts believe that contact with a loving, nurturing parent or caregiver can help children weather stress and reduce chances for lasting ill effects. A recent U.S. study of toddlers from families living in poverty is an example. Youngsters who had a strong parent relationship had lower stress-hormone levels when they arrived at a clinic to get vaccinations than those without that parent buffer.\u003c/p>\n\u003cp>\u003cstrong>What's Toxic Stress? \u003c/strong>\u003c/p>\n\u003cp>Scientists increasingly believe that persistent stress, particularly when there’s no nurturing parent to help kids cope, can cause stress hormones and internal inflammatory markers to smolder at elevated levels, raising risks for later heart disease, diabetes and other health problems.\u003c/p>\n\u003cp>The science is not settled, but some studies also have shown that persistent stress may alter brain structure in regions affecting emotions and regulating behavior. Imaging studies have found these regions are smaller than usual in severely traumatized children.\u003c/p>\n\u003cp>Harvard University neuroscientist Charles Nelson says children younger than 3, with rapidly developing brains, are most at risk for the toxic effects of ongoing stress. In his imaging studies, school-aged Romanian orphans sent to live with foster families had brains with less gray matter than orphans sent to nurturing foster homes before age 2.\u003c/p>\n\u003cp>\u003cstrong>What About Migrant Children \u003c/strong>\u003c/p>\n\u003cp>Central American children arriving with their families at the southern U.S. border have already endured the trauma of leaving their homes, some after violence or other threats, and faced the additional trauma of an arduous journey north, Nelson said Wednesday. “That may increase their susceptibility to the hazards of separation at the border,” he said.\u003c/p>\n\u003cp>Nelson said the images he’s seen of U.S. detention centers housing children remind him of his research with Romanian children. In a recent research newsletter, Nelson wrote, “The lessons we learned then taught us that housing children in institution-like settings, with rotating shifts of caregivers and unfavorable ratios of caregivers to children, may cause severe and permanent damage to their minds and bodies.”\u003c/p>\n\u003cp>“This is what science has taught us about separation,” he said Wednesday.\u003c/p>\n\u003cp>\u003cstrong>What Doctors Say \u003c/strong>\u003c/p>\n\u003cp>In 2017, a year before the separation policy was announced, the American Academy of Pediatrics advised against holding migrant children in detention centers and said they should receive health care and treatment to “support their well-being.”\u003c/p>\n\u003cp>The \u003ca href=\"https://www.aap.org/en-us/about-the-aap/aap-press-room/Pages/AAPStatementOpposingBorderSecurityandImmigrationReformAct.aspx\">academy\u003c/a> and several mental health \u003ca href=\"http://www.apa.org/news/press/releases/2018/06/family-separation-policy.aspx\">groups\u003c/a> recently denounced the separation policy and cautiously supported last week’s executive order to overturn it. Tuesday’s order from a federal judge mandating that thousands of families be reunited within 30 days is an important step, the academy said in a joint statement with UNICEF USA, but it won’t undo the trauma separated children have already faced, said Academy President Dr. Colleen Kraft.\u003c/p>\n\u003cp>Even if they are eventually reunited, these families will need “trauma-informed” care to help them cope, Kraft said.\u003c/p>\n\u003cp>In their statement Wednesday, the academy and UNICEF USA said their organizations “stand ready to assist in reunifying families and supporting their needs. Pediatricians across the country are prepared to provide care for immigrant children as they enter our communities.”\u003c/p>\n\u003cp>Kraft attempted to enter a children’s shelter in McAllen, Texas on Wednesday to see what attempts were being made to reunite children and parents but said authorities turned her away. During an April visit to the same shelter, Kraft saw children who were “eerily quiet” and one who was crying inconsolably while attendants watched.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>“Staff in the room were not allowed to pick her up and hold her,” Kraft said. It’s that kind of absence of nurturing contact that experts think worsens the effects of traumatic stress.\u003c/p>\n\n\u003c/div>\u003c/p>",
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"content": "\u003cp>Even beforehand — before the compulsive writing and the bipolar diagnosis, before the niche medical practice and the best-selling book — Dr. Alice Flaherty stuck out. She had grown up beside a duckweed-filled pond in rural New Jersey, and by the time she was a young adult, she’d become a neuroscientist in a family of engineers, a theorist among doers.[contextly_sidebar id=\"sXNIfkgjQfMG5MJwfPfkWITgj41OQW8E\"]\u003c/p>\n\u003cp>When she came home during breaks at Harvard, her father would rib her. “He’d say, ‘Yo, you talk big about pure science now, but you’re going to end up an engineer just like the rest of us,’” she recalled. “And when I went to med school, he was like, ‘See? See?’ And it’s totally true. It’s like tinkering. You tinker with the patients. It’s so fun. I love fixing broken machines.”\u003c/p>\n\u003cp>Her neurology work at Massachusetts General Hospital involves plenty of gadgetry — she heads up the deep brain stimulation unit, and sometimes uses electroconvulsive therapy to help patients with depression or mania — but these days, that’s not the kind of tinkering that’s at the front of her mind.\u003c/p>\n\u003cp>Instead, she has been toying with the boundaries of illness itself. She likes seeing patients other doctors have given up on. Many have faced questions about whether they’re really as sick as they say. For all of them, getting the proper treatment — pills or infusions or electrical currents — depends on a kind of collaboration with Flaherty, a workshop in which motivations are re-examined, stories reshaped, turns of phrase redefined.\u003c/p>\n\u003cp>“These poor patients are typically seen as just not wanting to get better, and I got interested in that whole thing, like if you want to get better then you’re sick, if you don’t want to get better, then it’s a vice,” she said. “What was it about us — the caregivers, family members, and doctors — what was it that made us attribute willfulness to people who were obviously miserable?”\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>Her schedule is stacked with examples.\u003c/p>\n\u003cp>There was the Parkinson’s patient who was able to move when playing with grandchildren but not when asked to take out the trash. “The spouse is like, ‘Bullshit, you’re just not trying,’ and that’s totally true. … They lack dopamine, which is very important for motivation,” Flaherty said.\u003c/p>\n\u003cp>There was the medical student who’d become catatonic when faced with an exam, and was accused of wanting, at some level, to jettison his career.[contextly_sidebar id=\"FisPQhjLXG17zQFS91fSEnl0b6zkrL5D\"]\u003c/p>\n\u003cp>There was the woman who came in with an anxiety-related tremor but insisted that, no, these shakes weren’t psychological. “You have to convince them, yes, you really are sick, I understand that, or they’ll never trust you,” Flaherty said. “And they are really sick, they’re disabled, totally.” Then, with a careful tweaking of language, she was able to prescribe the Valium that the patient had been refusing from other doctors, and the tremor faded.\u003c/p>\n\u003cp>To some, she might sound like a shrink tucked away inside a movement disorder clinic, and that isn’t entirely wrong. Historically, psychiatrists and neurologists often kept to their own floors, as if the feeling-thinking brain and the physical brain were two different organs. Flaherty was merging both long before it became a trend, explained Dr. Jerrold Rosenbaum, Mass. General’s psychiatrist-in-chief.\u003c/p>\n\u003cp>“When we had a complicated patient … and we needed a neurologist who understood how we think, Alice would be our go-to consultant. She’s a great bridge,” he said. “She’s more sophisticated with the use of our drugs than many of us are.”\u003c/p>\n\u003cp>But that doesn’t quite cover Flaherty’s unique role in the biome of Boston medicine. When the novelist William Styron was weighed down by depression, movement issues, and delusions — he thought that his writing hand had gone dead and his head was shrinking to the size of a pin — he sought out Flaherty.\u003c/p>\n\u003cp>She makes a habit of befriending her patients and counseling her friends. She sometimes worries her colleagues might see her as too empathetic, too credulous, too boundary-breaking. Then again, she knows from her own experience with mental illness that the opposite — an excess of formality, stiffness, distrust — can be worse.\u003c/p>\n\u003cp>Flaherty had heard about Stephanie Zaia before they’d ever met. Clinicians talk, and a case like Zaia’s made them talk more than usual. Her symptoms seemed so strange, and so complicated, that they were picked apart during grand rounds, at medical meetings, and in hospital staff rooms. What struck Flaherty was that her colleagues seemed to suggest that Zaia had invented her own bodily inferno.\u003c/p>\n\u003cp>The trouble began when Zaia was a 14-year-old in Medfield, Mass. She was a competitive swimmer, obsessed enough to make her parents drive her to practices at 5:30 a.m. She swam every weekday, spent her weekends at meets, but all of a sudden, at the end of a race in May 2003, her body went limp and she couldn’t get out of the pool.[contextly_sidebar id=\"YgnHkwDfRuaRza68Zle6mgr7qAhNQXhh\"]\u003c/p>\n\u003cp>Then she began having trouble moving elsewhere, too. Walking became hard. Sometimes she struggled to breathe. She stopped being able to digest, vomiting up almost everything she ate. She began to shed weight that she couldn’t afford to lose.\u003c/p>\n\u003cp>Her parents brought her to neurologists and psychiatrists and specialists of the gut, bouncing from practice to practice, from Massachusetts all the way to Maryland. They kept telling Zaia the same thing again and again: “They’d say, ‘Oh, it’s all in your head,” she said.\u003c/p>\n\u003cp>At first, Zaia and her parents went along with the idea. They probed her past for possible instances of trauma, but could find nothing besides garden-variety middle-school meanness from other kids, which hadn’t gone on for very long anyway. And the suggestions of depression and anorexia didn’t seem to add up with her mounting difficulty to move. “‘You’re trying not to walk because you hate to swim.’ That’s what they said,” Zaia recalled. “They were just like, ‘It’s a conversion disorder: You’re converting your not wanting to do this into physical symptoms.’”\u003c/p>\n\u003cp>No matter what she said, she couldn’t convince them that she wanted nothing more than to be able to swim again. Nor could she convince them, when they claimed her muscle spasms were an embodiment of her jealousy toward her siblings, that she did not feel jealous.\u003c/p>\n\u003cp>Her medical file began to take on an authority of its own, as if the hypothesis that her illness was psychological had, through repetition, become a fact. Doctors couldn’t unearth some underlying cause for the muscle tightness, and could find no relation to the mutiny in her gut. But there, in the pages of her record, was an explanation capable of tying these disparate threads together. The symptoms didn’t make sense, they thought, because she was, in some subconscious stratum, making them up.\u003c/p>\n\u003cp>It was only after years, in 2006, that Zaia got a diagnosis of primary dystonia — a condition characterized by involuntary muscle contractions — and learned just how common this kind of accusation is. “I’ve met lots of patients who spend five years being misdiagnosed,” said Pamela Sloate, a patient activist and board member of the Dystonia Medical Research Foundation. “They don’t have the skills to diagnose dystonia, so they tell the patient it’s imagined, or that it’s caused by depression.”\u003c/p>\n\u003cp>That can have a profound effect. As Zaia put it, “When somebody tells you that enough times, you start to believe it.”\u003c/p>\n\u003cp>Self-blame is something that Flaherty is familiar with. She felt it acutely in 1998, when her twin boys died immediately after birth. They were premature, so tiny their hands could hardly fit around her finger. To bury their ashes, she secretly took a folding shovel into Mount Auburn Cemetery to look for a spot where no one would see her digging. She chose a patch of shrubbery beside a pond grown green with duckweed: It reminded her of the scummy pool back home.[contextly_sidebar id=\"rhuJ5KiqdPloHZe58oqJ8GhRRj1a2q7n\"]\u003c/p>\n\u003cp>Ten days after their death, her sadness morphed into an overwhelming desire to write. She wrote on everything: paper, napkins, computers, her own skin. She’d written plenty before — waking up early to write a neurology handbook while she was a resident — but now it was uncontrollable, and the style had changed. “Looking at this stuff, I’m like, ‘Oh my, God, this is like teen diary garbage,’” she said.\u003c/p>\n\u003cp>She bounced between mania and depression, becoming obsessed with the idea that she’d been a bad mother, that it was all her fault. She knew, on the one hand, that she was sick — she’d been newly diagnosed with bipolar disorder, and was taking a pharmacopeia of pills — but also felt that her illness wasn’t real, that she was just fishing for attention.\u003c/p>\n\u003cp>“The most painful part of it was I thought I was making it up,” she said. “I thought I was this total loser that was making up something that had me in the hospital for nine days.”\u003c/p>\n\u003cp>Sometimes, she couldn’t physically lift her hand to her mouth to take her meds. She’d rock her arm back and forth, coaxing herself, like a volleyball player preparing to make a serve.\u003c/p>\n\u003cp>She was consumed by the doublethink of depression. She knew her symptoms weren’t fictional, but what if they were all in her head? She knew her obstetrician didn’t hate her, but what if he did? She wanted him to say that something terrible had happened. She wanted him to echo her own distress. She wanted him to cry.\u003c/p>\n\u003cp>Flaherty suspected the episode would end her medical career. Her colleagues told her not to tell anyone, but she was manic, and told everyone. She wrote about her illness in her book “The Midnight Disease,” and her story wound up in the glossy pages of magazines. Patients could idly flip through her postpartum mania while waiting for their appointments.\u003c/p>\n\u003cp>“The people who are most afraid of mental illness are doctors,” Flaherty said. “It turned out my patients were fine with it. … One guy said, ‘Yeah, that manic depressive thing you have, my internist has that. … Every six months or so, they have to lock him up, because he runs down the middle of the street naked. But I stay with him because he’s a really good doctor when he’s not crazy.’”[contextly_sidebar id=\"AQvxss0iAWMjp5nhtNV4d3hmbJrV2oIX\"]\u003c/p>\n\u003cp>Even as her mood stabilizers did their job, the impressions she had during illness stayed vivid: her conviction that she’d created her own symptoms, her over-analysis of her obstetrician’s veneer.\u003c/p>\n\u003cp>“She knows from experience how imperious doctors can be. … When you’re really in a lot of pain, or not sleeping, or whatever, in an acute phase, you feel like a supplicant,” said journalist and author Pagan Kennedy. The two had met in a local writers’ group, and when Kennedy experienced a mysterious bout of insomnia and acute pain, Flaherty counseled her not to rush into surgery, and checked in with her every day, listening. Kennedy is now making a podcast about Flaherty’s treatment of Styron.\u003c/p>\n\u003cp>The same skills that cement a friendship, Flaherty has found, are useful clinical tools. But bedside manner hasn’t come naturally to her. “I didn’t have any body language,” she said. “I was brought up in this WASP community with 500 guns in the basement. … We communicated by raising our eyebrows one teeny little tiny bit.”\u003c/p>\n\u003cp>So she sometimes glances at a mirror hidden in among the drawings and plants of her office, checking her own features, making sure she echoes the patient’s devastation or anger or joy. Above all — whether the symptoms are psychological, physical, or some combination of the two — she wants them to feel heard. By now, after years of practice, she says that most of the emotions behind her gestures are real.\u003c/p>\n\u003cp>When Flaherty finally met her, in the fall of 2012, Zaia had been sick for almost 10 years. She needed a wheelchair, and her muscles were so tight she was not able to sit up. Every so often, she’d get dystonic storms: Her back would arch so she couldn’t breathe, her neck yanked backwards, her arms pinned behind her, her legs pulled as far as they would go. “I would turn into a literal pretzel,” she said.[contextly_sidebar id=\"IBjHyTyOkXKgQvZHUH9PFd3FV0V9LNU5\"]\u003c/p>\n\u003cp>The relationship began as pure coincidence. Zaia was an inpatient at Mass. General, home sick from the University of Illinois, Urbana-Champaign, and Flaherty happened to be the neurologist on duty. It wasn’t just the medical crisis that was worrying Zaia’s parents. Her primary neurologist, who had been treating Zaia’s dystonia for years, had put a letter in the medical record that signaled a change of tune.\u003c/p>\n\u003cp>“She was basically saying she had never believed Steph in the first place,” said her mother, Diane. “She basically treated Stephanie for dystonia for … years, and then said she did not have dystonia, and said that she was a wacko.”\u003c/p>\n\u003cp>And so Flaherty agreed to be Zaia’s neurologist. Already, to the family, that was a minor miracle. “No one wanted to touch me,” said Zaia.\u003c/p>\n\u003cp>During those years, Zaia had thought she might end up bed-bound in a nursing home. Now, she’s up every morning at 5:15 to catch the 7:16 train from Dedham into South Station. Her work, at Easter Seals Massachusetts, a nonprofit that provides disability services, is only a block away. She also helps run her own organization, PATH-WAY, which puts together social gatherings accessible to everyone and anyone, no matter their physical ability. To find members, she went to support groups for illnesses she didn’t have. At the end of the day, she catches the 4:43 back to Dedham.\u003c/p>\n\u003cp>[ad floatright]\u003c/p>\n\u003cp>\u003cem>This\u003ca href=\"https://www.statnews.com/2018/06/19/alice-flaherty-mass-general-neurologist/\" target=\"_blank\" rel=\"noopener\"> story\u003c/a> was originally published by \u003ca href=\"https://www.statnews.com/2018/06/19/alice-flaherty-mass-general-neurologist/\" target=\"_blank\" rel=\"noopener\">STAT\u003c/a>, an online publication of Boston Globe Media that covers health, medicine, and scientific discovery.\u003c/em>\u003c/p>\n\n",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp>Even beforehand — before the compulsive writing and the bipolar diagnosis, before the niche medical practice and the best-selling book — Dr. Alice Flaherty stuck out. She had grown up beside a duckweed-filled pond in rural New Jersey, and by the time she was a young adult, she’d become a neuroscientist in a family of engineers, a theorist among doers.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>When she came home during breaks at Harvard, her father would rib her. “He’d say, ‘Yo, you talk big about pure science now, but you’re going to end up an engineer just like the rest of us,’” she recalled. “And when I went to med school, he was like, ‘See? See?’ And it’s totally true. It’s like tinkering. You tinker with the patients. It’s so fun. I love fixing broken machines.”\u003c/p>\n\u003cp>Her neurology work at Massachusetts General Hospital involves plenty of gadgetry — she heads up the deep brain stimulation unit, and sometimes uses electroconvulsive therapy to help patients with depression or mania — but these days, that’s not the kind of tinkering that’s at the front of her mind.\u003c/p>\n\u003cp>Instead, she has been toying with the boundaries of illness itself. She likes seeing patients other doctors have given up on. Many have faced questions about whether they’re really as sick as they say. For all of them, getting the proper treatment — pills or infusions or electrical currents — depends on a kind of collaboration with Flaherty, a workshop in which motivations are re-examined, stories reshaped, turns of phrase redefined.\u003c/p>\n\u003cp>“These poor patients are typically seen as just not wanting to get better, and I got interested in that whole thing, like if you want to get better then you’re sick, if you don’t want to get better, then it’s a vice,” she said. “What was it about us — the caregivers, family members, and doctors — what was it that made us attribute willfulness to people who were obviously miserable?”\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>Her schedule is stacked with examples.\u003c/p>\n\u003cp>There was the Parkinson’s patient who was able to move when playing with grandchildren but not when asked to take out the trash. “The spouse is like, ‘Bullshit, you’re just not trying,’ and that’s totally true. … They lack dopamine, which is very important for motivation,” Flaherty said.\u003c/p>\n\u003cp>There was the medical student who’d become catatonic when faced with an exam, and was accused of wanting, at some level, to jettison his career.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>There was the woman who came in with an anxiety-related tremor but insisted that, no, these shakes weren’t psychological. “You have to convince them, yes, you really are sick, I understand that, or they’ll never trust you,” Flaherty said. “And they are really sick, they’re disabled, totally.” Then, with a careful tweaking of language, she was able to prescribe the Valium that the patient had been refusing from other doctors, and the tremor faded.\u003c/p>\n\u003cp>To some, she might sound like a shrink tucked away inside a movement disorder clinic, and that isn’t entirely wrong. Historically, psychiatrists and neurologists often kept to their own floors, as if the feeling-thinking brain and the physical brain were two different organs. Flaherty was merging both long before it became a trend, explained Dr. Jerrold Rosenbaum, Mass. General’s psychiatrist-in-chief.\u003c/p>\n\u003cp>“When we had a complicated patient … and we needed a neurologist who understood how we think, Alice would be our go-to consultant. She’s a great bridge,” he said. “She’s more sophisticated with the use of our drugs than many of us are.”\u003c/p>\n\u003cp>But that doesn’t quite cover Flaherty’s unique role in the biome of Boston medicine. When the novelist William Styron was weighed down by depression, movement issues, and delusions — he thought that his writing hand had gone dead and his head was shrinking to the size of a pin — he sought out Flaherty.\u003c/p>\n\u003cp>She makes a habit of befriending her patients and counseling her friends. She sometimes worries her colleagues might see her as too empathetic, too credulous, too boundary-breaking. Then again, she knows from her own experience with mental illness that the opposite — an excess of formality, stiffness, distrust — can be worse.\u003c/p>\n\u003cp>Flaherty had heard about Stephanie Zaia before they’d ever met. Clinicians talk, and a case like Zaia’s made them talk more than usual. Her symptoms seemed so strange, and so complicated, that they were picked apart during grand rounds, at medical meetings, and in hospital staff rooms. What struck Flaherty was that her colleagues seemed to suggest that Zaia had invented her own bodily inferno.\u003c/p>\n\u003cp>The trouble began when Zaia was a 14-year-old in Medfield, Mass. She was a competitive swimmer, obsessed enough to make her parents drive her to practices at 5:30 a.m. She swam every weekday, spent her weekends at meets, but all of a sudden, at the end of a race in May 2003, her body went limp and she couldn’t get out of the pool.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>Then she began having trouble moving elsewhere, too. Walking became hard. Sometimes she struggled to breathe. She stopped being able to digest, vomiting up almost everything she ate. She began to shed weight that she couldn’t afford to lose.\u003c/p>\n\u003cp>Her parents brought her to neurologists and psychiatrists and specialists of the gut, bouncing from practice to practice, from Massachusetts all the way to Maryland. They kept telling Zaia the same thing again and again: “They’d say, ‘Oh, it’s all in your head,” she said.\u003c/p>\n\u003cp>At first, Zaia and her parents went along with the idea. They probed her past for possible instances of trauma, but could find nothing besides garden-variety middle-school meanness from other kids, which hadn’t gone on for very long anyway. And the suggestions of depression and anorexia didn’t seem to add up with her mounting difficulty to move. “‘You’re trying not to walk because you hate to swim.’ That’s what they said,” Zaia recalled. “They were just like, ‘It’s a conversion disorder: You’re converting your not wanting to do this into physical symptoms.’”\u003c/p>\n\u003cp>No matter what she said, she couldn’t convince them that she wanted nothing more than to be able to swim again. Nor could she convince them, when they claimed her muscle spasms were an embodiment of her jealousy toward her siblings, that she did not feel jealous.\u003c/p>\n\u003cp>Her medical file began to take on an authority of its own, as if the hypothesis that her illness was psychological had, through repetition, become a fact. Doctors couldn’t unearth some underlying cause for the muscle tightness, and could find no relation to the mutiny in her gut. But there, in the pages of her record, was an explanation capable of tying these disparate threads together. The symptoms didn’t make sense, they thought, because she was, in some subconscious stratum, making them up.\u003c/p>\n\u003cp>It was only after years, in 2006, that Zaia got a diagnosis of primary dystonia — a condition characterized by involuntary muscle contractions — and learned just how common this kind of accusation is. “I’ve met lots of patients who spend five years being misdiagnosed,” said Pamela Sloate, a patient activist and board member of the Dystonia Medical Research Foundation. “They don’t have the skills to diagnose dystonia, so they tell the patient it’s imagined, or that it’s caused by depression.”\u003c/p>\n\u003cp>That can have a profound effect. As Zaia put it, “When somebody tells you that enough times, you start to believe it.”\u003c/p>\n\u003cp>Self-blame is something that Flaherty is familiar with. She felt it acutely in 1998, when her twin boys died immediately after birth. They were premature, so tiny their hands could hardly fit around her finger. To bury their ashes, she secretly took a folding shovel into Mount Auburn Cemetery to look for a spot where no one would see her digging. She chose a patch of shrubbery beside a pond grown green with duckweed: It reminded her of the scummy pool back home.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>Ten days after their death, her sadness morphed into an overwhelming desire to write. She wrote on everything: paper, napkins, computers, her own skin. She’d written plenty before — waking up early to write a neurology handbook while she was a resident — but now it was uncontrollable, and the style had changed. “Looking at this stuff, I’m like, ‘Oh my, God, this is like teen diary garbage,’” she said.\u003c/p>\n\u003cp>She bounced between mania and depression, becoming obsessed with the idea that she’d been a bad mother, that it was all her fault. She knew, on the one hand, that she was sick — she’d been newly diagnosed with bipolar disorder, and was taking a pharmacopeia of pills — but also felt that her illness wasn’t real, that she was just fishing for attention.\u003c/p>\n\u003cp>“The most painful part of it was I thought I was making it up,” she said. “I thought I was this total loser that was making up something that had me in the hospital for nine days.”\u003c/p>\n\u003cp>Sometimes, she couldn’t physically lift her hand to her mouth to take her meds. She’d rock her arm back and forth, coaxing herself, like a volleyball player preparing to make a serve.\u003c/p>\n\u003cp>She was consumed by the doublethink of depression. She knew her symptoms weren’t fictional, but what if they were all in her head? She knew her obstetrician didn’t hate her, but what if he did? She wanted him to say that something terrible had happened. She wanted him to echo her own distress. She wanted him to cry.\u003c/p>\n\u003cp>Flaherty suspected the episode would end her medical career. Her colleagues told her not to tell anyone, but she was manic, and told everyone. She wrote about her illness in her book “The Midnight Disease,” and her story wound up in the glossy pages of magazines. Patients could idly flip through her postpartum mania while waiting for their appointments.\u003c/p>\n\u003cp>“The people who are most afraid of mental illness are doctors,” Flaherty said. “It turned out my patients were fine with it. … One guy said, ‘Yeah, that manic depressive thing you have, my internist has that. … Every six months or so, they have to lock him up, because he runs down the middle of the street naked. But I stay with him because he’s a really good doctor when he’s not crazy.’”\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>Even as her mood stabilizers did their job, the impressions she had during illness stayed vivid: her conviction that she’d created her own symptoms, her over-analysis of her obstetrician’s veneer.\u003c/p>\n\u003cp>“She knows from experience how imperious doctors can be. … When you’re really in a lot of pain, or not sleeping, or whatever, in an acute phase, you feel like a supplicant,” said journalist and author Pagan Kennedy. The two had met in a local writers’ group, and when Kennedy experienced a mysterious bout of insomnia and acute pain, Flaherty counseled her not to rush into surgery, and checked in with her every day, listening. Kennedy is now making a podcast about Flaherty’s treatment of Styron.\u003c/p>\n\u003cp>The same skills that cement a friendship, Flaherty has found, are useful clinical tools. But bedside manner hasn’t come naturally to her. “I didn’t have any body language,” she said. “I was brought up in this WASP community with 500 guns in the basement. … We communicated by raising our eyebrows one teeny little tiny bit.”\u003c/p>\n\u003cp>So she sometimes glances at a mirror hidden in among the drawings and plants of her office, checking her own features, making sure she echoes the patient’s devastation or anger or joy. Above all — whether the symptoms are psychological, physical, or some combination of the two — she wants them to feel heard. By now, after years of practice, she says that most of the emotions behind her gestures are real.\u003c/p>\n\u003cp>When Flaherty finally met her, in the fall of 2012, Zaia had been sick for almost 10 years. She needed a wheelchair, and her muscles were so tight she was not able to sit up. Every so often, she’d get dystonic storms: Her back would arch so she couldn’t breathe, her neck yanked backwards, her arms pinned behind her, her legs pulled as far as they would go. “I would turn into a literal pretzel,” she said.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>The relationship began as pure coincidence. Zaia was an inpatient at Mass. General, home sick from the University of Illinois, Urbana-Champaign, and Flaherty happened to be the neurologist on duty. It wasn’t just the medical crisis that was worrying Zaia’s parents. Her primary neurologist, who had been treating Zaia’s dystonia for years, had put a letter in the medical record that signaled a change of tune.\u003c/p>\n\u003cp>“She was basically saying she had never believed Steph in the first place,” said her mother, Diane. “She basically treated Stephanie for dystonia for … years, and then said she did not have dystonia, and said that she was a wacko.”\u003c/p>\n\u003cp>And so Flaherty agreed to be Zaia’s neurologist. Already, to the family, that was a minor miracle. “No one wanted to touch me,” said Zaia.\u003c/p>\n\u003cp>During those years, Zaia had thought she might end up bed-bound in a nursing home. Now, she’s up every morning at 5:15 to catch the 7:16 train from Dedham into South Station. Her work, at Easter Seals Massachusetts, a nonprofit that provides disability services, is only a block away. She also helps run her own organization, PATH-WAY, which puts together social gatherings accessible to everyone and anyone, no matter their physical ability. To find members, she went to support groups for illnesses she didn’t have. At the end of the day, she catches the 4:43 back to Dedham.\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>\u003cem>This\u003ca href=\"https://www.statnews.com/2018/06/19/alice-flaherty-mass-general-neurologist/\" target=\"_blank\" rel=\"noopener\"> story\u003c/a> was originally published by \u003ca href=\"https://www.statnews.com/2018/06/19/alice-flaherty-mass-general-neurologist/\" target=\"_blank\" rel=\"noopener\">STAT\u003c/a>, an online publication of Boston Globe Media that covers health, medicine, and scientific discovery.\u003c/em>\u003c/p>\n\n\u003c/div>\u003c/p>",
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"disqusTitle": "Stanford Pioneer in Brain Research Wins Prestigious Kyoto Prize",
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"content": "\u003cp>Karl Deisseroth, a Stanford University professor of bioengineering and psychiatry, has been awarded an international prize worth more than $900,000 for his work in illuminating brain activity with light. The exact amount of the prize was 100 million yen.\u003c/p>\n\u003caside class=\"pullquote alignright\">'Karl has created a revolutionary technology that has broadened our understanding of brain disorders and may one day yield treatments to the millions with these disorders.'\u003ccite>Lloyd Minor, Stanford School of Medicine\u003c/cite>\u003c/aside>\n\u003cp>Deisseroth is receiving the 2018 \u003ca href=\"https://www.kyotoprize.org/en/\" target=\"_blank\" rel=\"noopener\">Kyoto Prize\u003c/a> for advanced technology. Awards are also granted for basic sciences, and arts and philosophy. Some of its recipients have gone on to win the Nobel Prize.\u003c/p>\n\u003cp>\u003ca href=\"https://web.stanford.edu/group/dlab/about_pi.html\" target=\"_blank\" rel=\"noopener\">Deisseroth,\u003c/a> the youngest person to ever receive the prize, is being honored for developing \u003ca href=\"http://web.stanford.edu/group/dlab/optogenetics/\">optogenetics\u003c/a>, a technology that uses light-sensitive proteins to manipulate brain cell activity. The method allows scientists to glean more information about brain disorders and to probe how the nervous system works.\u003c/p>\n\u003cp>\"It is humbling to see all the distinguished people who have won the Kyoto Prize, and to be listed with them. It is wonderful to see pure basic science discoveries recognized in this way,\" said Deisseroth.\u003c/p>\n\u003cp>The technique developed by Deisseroth and his team manipulates cellular activity by delivering a pulse of light to a particular cell through an optical fiber that has been implanted in the animal’s brain. The method yields information on how brain cells give rise to sensations, memories and actions.\u003c/p>\n\u003cfigure id=\"attachment_442697\" class=\"wp-caption alignright\" style=\"max-width: 480px\">\u003cimg class=\"size-full wp-image-442697\" src=\"https://ww2.kqed.org/futureofyou/wp-content/uploads/sites/13/2018/06/image01.jpg\" alt=\"\" width=\"480\" height=\"360\" srcset=\"https://ww2.kqed.org/app/uploads/sites/13/2018/06/image01.jpg 480w, https://ww2.kqed.org/app/uploads/sites/13/2018/06/image01-160x120.jpg 160w, https://ww2.kqed.org/app/uploads/sites/13/2018/06/image01-240x180.jpg 240w, https://ww2.kqed.org/app/uploads/sites/13/2018/06/image01-375x281.jpg 375w\" sizes=\"(max-width: 480px) 100vw, 480px\">\u003cfigcaption class=\"wp-caption-text\">The Kyoto prize medal. \u003ccite>(Inamori Foundation)\u003c/cite>\u003c/figcaption>\u003c/figure>\n\u003cp>“A brilliant and innovative investigator, Karl has created a revolutionary technology that has broadened our understanding of brain disorders and may one day yield treatments to the millions with these disorders,” said Dr. Lloyd Minor, dean of the \u003ca href=\"http://med.stanford.edu/\" target=\"_blank\" rel=\"noopener\">Stanford School of Medicine\u003c/a>, in a statement. “His receipt of the Kyoto Prize is inordinately well-deserved and the product of his unmatched scientific vision.\"\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>Deisseroth has said his research\u003ca href=\"https://www.scientificamerican.com/article/optogenetics-controlling/\" target=\"_blank\" rel=\"noopener\"> was motivated by\u003c/a> a desire to better understand psychiatric disorders, the leading cause of years of life lost to death or disability.\u003c/p>\n\u003cp>Deisseroth’s research, developed between 2004 and 2009, is used by laboratories around the world.\u003c/p>\n\u003cp>\"Thousands of laboratories around the world have used optogenetics to satisfy their own curiosity about the natural world, and to test ideas in a broad range of systems and species,\" he said. \"Many people are also studying brain diseases, and optogenetics-guided clinical trials have already started with some success, bringing a new kind of hope to people affected by these diseases such as substance dependence and depression.\"\u003c/p>\n\u003cp>\u003ciframe width=\"640\" height=\"360\" src=\"https://www.youtube.com/embed/CE6uNFFZ0qc?feature=oembed\" frameborder=\"0\" allow=\"accelerometer; autoplay; encrypted-media; gyroscope; picture-in-picture\" allowfullscreen>\u003c/iframe>\u003c/p>\n\u003cp>What makes optogenetics so groundbreaking is its level of precision in controlling defined activity within specific cells, according to I-Han Chou, senior editor at the journal \"Nature\".[contextly_sidebar id=\"08EVHeybNXS0fTuglKI55A1KVy7EFUpl\"]\u003c/p>\n\u003cp>\"If you imagine the brain as this city, up until now we have been looking at it as if from space,\" Chou\u003ca href=\"https://www.weforum.org/agenda/2016/10/what-is-optogenetics/\" target=\"_blank\" rel=\"noopener\"> told the\u003c/a> World Economic Forum site. \"We haven't had the tools to do anything beyond seeing what the whole city block is doing. What you actually want to know is what the individual components of the city are, what the people are doing, and what's the information being transported from one part of the city to another.\"\u003c/p>\n\u003cp>The technique could transform how neurological disorders such as Parkinson's disease are treated.\u003c/p>\n\u003cp>\"When things go wrong with the brain it is just so devastating. One of the hopes for optogenetics is that if it can work in humans, it might be used as a tool for restoring brain function,\" Chou added.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp> \u003c/p>\n\n",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp>Karl Deisseroth, a Stanford University professor of bioengineering and psychiatry, has been awarded an international prize worth more than $900,000 for his work in illuminating brain activity with light. The exact amount of the prize was 100 million yen.\u003c/p>\n\u003caside class=\"pullquote alignright\">'Karl has created a revolutionary technology that has broadened our understanding of brain disorders and may one day yield treatments to the millions with these disorders.'\u003ccite>Lloyd Minor, Stanford School of Medicine\u003c/cite>\u003c/aside>\n\u003cp>Deisseroth is receiving the 2018 \u003ca href=\"https://www.kyotoprize.org/en/\" target=\"_blank\" rel=\"noopener\">Kyoto Prize\u003c/a> for advanced technology. Awards are also granted for basic sciences, and arts and philosophy. Some of its recipients have gone on to win the Nobel Prize.\u003c/p>\n\u003cp>\u003ca href=\"https://web.stanford.edu/group/dlab/about_pi.html\" target=\"_blank\" rel=\"noopener\">Deisseroth,\u003c/a> the youngest person to ever receive the prize, is being honored for developing \u003ca href=\"http://web.stanford.edu/group/dlab/optogenetics/\">optogenetics\u003c/a>, a technology that uses light-sensitive proteins to manipulate brain cell activity. The method allows scientists to glean more information about brain disorders and to probe how the nervous system works.\u003c/p>\n\u003cp>\"It is humbling to see all the distinguished people who have won the Kyoto Prize, and to be listed with them. It is wonderful to see pure basic science discoveries recognized in this way,\" said Deisseroth.\u003c/p>\n\u003cp>The technique developed by Deisseroth and his team manipulates cellular activity by delivering a pulse of light to a particular cell through an optical fiber that has been implanted in the animal’s brain. The method yields information on how brain cells give rise to sensations, memories and actions.\u003c/p>\n\u003cfigure id=\"attachment_442697\" class=\"wp-caption alignright\" style=\"max-width: 480px\">\u003cimg class=\"size-full wp-image-442697\" src=\"https://ww2.kqed.org/futureofyou/wp-content/uploads/sites/13/2018/06/image01.jpg\" alt=\"\" width=\"480\" height=\"360\" srcset=\"https://ww2.kqed.org/app/uploads/sites/13/2018/06/image01.jpg 480w, https://ww2.kqed.org/app/uploads/sites/13/2018/06/image01-160x120.jpg 160w, https://ww2.kqed.org/app/uploads/sites/13/2018/06/image01-240x180.jpg 240w, https://ww2.kqed.org/app/uploads/sites/13/2018/06/image01-375x281.jpg 375w\" sizes=\"(max-width: 480px) 100vw, 480px\">\u003cfigcaption class=\"wp-caption-text\">The Kyoto prize medal. \u003ccite>(Inamori Foundation)\u003c/cite>\u003c/figcaption>\u003c/figure>\n\u003cp>“A brilliant and innovative investigator, Karl has created a revolutionary technology that has broadened our understanding of brain disorders and may one day yield treatments to the millions with these disorders,” said Dr. Lloyd Minor, dean of the \u003ca href=\"http://med.stanford.edu/\" target=\"_blank\" rel=\"noopener\">Stanford School of Medicine\u003c/a>, in a statement. “His receipt of the Kyoto Prize is inordinately well-deserved and the product of his unmatched scientific vision.\"\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>Deisseroth has said his research\u003ca href=\"https://www.scientificamerican.com/article/optogenetics-controlling/\" target=\"_blank\" rel=\"noopener\"> was motivated by\u003c/a> a desire to better understand psychiatric disorders, the leading cause of years of life lost to death or disability.\u003c/p>\n\u003cp>Deisseroth’s research, developed between 2004 and 2009, is used by laboratories around the world.\u003c/p>\n\u003cp>\"Thousands of laboratories around the world have used optogenetics to satisfy their own curiosity about the natural world, and to test ideas in a broad range of systems and species,\" he said. \"Many people are also studying brain diseases, and optogenetics-guided clinical trials have already started with some success, bringing a new kind of hope to people affected by these diseases such as substance dependence and depression.\"\u003c/p>\n\u003cp>\u003ciframe width=\"640\" height=\"360\" src=\"https://www.youtube.com/embed/CE6uNFFZ0qc?feature=oembed\" frameborder=\"0\" allow=\"accelerometer; autoplay; encrypted-media; gyroscope; picture-in-picture\" allowfullscreen>\u003c/iframe>\u003c/p>\n\u003cp>What makes optogenetics so groundbreaking is its level of precision in controlling defined activity within specific cells, according to I-Han Chou, senior editor at the journal \"Nature\".\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>\"If you imagine the brain as this city, up until now we have been looking at it as if from space,\" Chou\u003ca href=\"https://www.weforum.org/agenda/2016/10/what-is-optogenetics/\" target=\"_blank\" rel=\"noopener\"> told the\u003c/a> World Economic Forum site. \"We haven't had the tools to do anything beyond seeing what the whole city block is doing. What you actually want to know is what the individual components of the city are, what the people are doing, and what's the information being transported from one part of the city to another.\"\u003c/p>\n\u003cp>The technique could transform how neurological disorders such as Parkinson's disease are treated.\u003c/p>\n\u003cp>\"When things go wrong with the brain it is just so devastating. One of the hopes for optogenetics is that if it can work in humans, it might be used as a tool for restoring brain function,\" Chou added.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp> \u003c/p>\n\n\u003c/div>\u003c/p>",
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"disqusTitle": "A New Look At Why Our Brains Evolved To Be So Big",
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"content": "\u003cp>Why do people have such big brains? Some researchers asked a really powerful brain — a computer — and got back a surprising answer.[contextly_sidebar id=\"d0DIuOWUOwVJhoxMb7LQpHdQ7uLJlr7Q\"]\u003c/p>\n\u003cp>In relation to body size, our brains are huge, about six times larger than one would expect from other mammals. And this three-pound organ sucks up fully 20 percent of the body’s energy needs. Evolutionary theory says to build and maintain something that costly, there must have been a very good reason.\u003c/p>\n\u003cp>But what is it? Did our ancestors need more brainpower to cope with the environment, such as finding and storing food? Or was it driven more by the social complexities of dealing with their peers? Or was it all about the challenge of learning and teaching cultural knowledge?\u003c/p>\n\u003cp>In the past, scientists have tried to tease out an answer mostly by analyzing correlations, like how brain size relates to the size of social groups in living apes and our fossil ancestors.\u003c/p>\n\u003cp>Mauricio Gonzalez-Forero and Andy Gardner of St. Andrews University in Scotland turned instead to computer simulations. They report the result in a paper released Wednesday by the journal Nature.\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>The researchers created a hypothetical population of females, focusing on just one sex for simplicity, and followed them as they faced the challenges of living. The researchers plugged in data on things like a newborn’s brain size and the energy costs of the brain and reproductive organs. And they simulated tasks that resemble the environmental and social challenges included in theories about brain evolution.[contextly_sidebar id=\"BoHBcN3FhLEeIu2K51PXBnKupNSL1pYA\"]\u003c/p>\n\u003cp>The computer pondered how the pressures of each challenge might affect changes of brain size over time. Drawing on evolutionary theory, it calculated how much energy the females would be expected to invest in growing the brain versus other tissues under the different challenges. It found that stronger mental demands tended to produce bigger brains.\u003c/p>\n\u003cp>Gonzalez-Forero said he and Gardner expected a strong contribution to bigger brains from social challenges, which he said has been the favorite idea for decades.\u003c/p>\n\u003cp>But their surprise, the computer said about 60 percent of the effect on boosting brain size came from an individual dealing with the environment on one’s own, as in finding, storing and cooking food, and making stone tools. Another 30 percent came from cooperating to deal with the environment, such as banding together to hunt. The final 10 percent from competition between groups of people.\u003c/p>\n\u003cp>While the study didn’t specifically look at the impact of cultural tasks, it gave evidence of a substantial influence from them, too, Gonzalez-Forero said. He plans to assess cultural factors in the future.\u003c/p>\n\u003cp>In any case, the results are not intended to be the final word, but rather an encouragement for other researchers to use computer simulation for studying the question of brain size. Much exciting work remains to be done, he said.\u003c/p>\n\u003cp>Experts unconnected to the study were skeptical of the conclusions.\u003c/p>\n\u003cp>Dean Falk, a brain-evolution expert at Florida State University, said the work doesn’t assess the longstanding hypothesis that the development of language may have driven expansion of the brain. Gonzalez-Forero said the language idea is to some extent part of the cultural factors that remain to be addressed.\u003c/p>\n\u003cp>Falk also said the simulation strategy may have overemphasized the role of energy demands in influencing brain growth.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>Robin Dunbar, a professor of evolutionary psychology at Oxford University, and paleoanthropologist Richard Potts of the Smithsonian Institution said they didn’t think the simulation adequately mimicked the lives our ancestors lived.\u003c/p>\n\n",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp>Why do people have such big brains? Some researchers asked a really powerful brain — a computer — and got back a surprising answer.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>In relation to body size, our brains are huge, about six times larger than one would expect from other mammals. And this three-pound organ sucks up fully 20 percent of the body’s energy needs. Evolutionary theory says to build and maintain something that costly, there must have been a very good reason.\u003c/p>\n\u003cp>But what is it? Did our ancestors need more brainpower to cope with the environment, such as finding and storing food? Or was it driven more by the social complexities of dealing with their peers? Or was it all about the challenge of learning and teaching cultural knowledge?\u003c/p>\n\u003cp>In the past, scientists have tried to tease out an answer mostly by analyzing correlations, like how brain size relates to the size of social groups in living apes and our fossil ancestors.\u003c/p>\n\u003cp>Mauricio Gonzalez-Forero and Andy Gardner of St. Andrews University in Scotland turned instead to computer simulations. They report the result in a paper released Wednesday by the journal Nature.\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>The researchers created a hypothetical population of females, focusing on just one sex for simplicity, and followed them as they faced the challenges of living. The researchers plugged in data on things like a newborn’s brain size and the energy costs of the brain and reproductive organs. And they simulated tasks that resemble the environmental and social challenges included in theories about brain evolution.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>The computer pondered how the pressures of each challenge might affect changes of brain size over time. Drawing on evolutionary theory, it calculated how much energy the females would be expected to invest in growing the brain versus other tissues under the different challenges. It found that stronger mental demands tended to produce bigger brains.\u003c/p>\n\u003cp>Gonzalez-Forero said he and Gardner expected a strong contribution to bigger brains from social challenges, which he said has been the favorite idea for decades.\u003c/p>\n\u003cp>But their surprise, the computer said about 60 percent of the effect on boosting brain size came from an individual dealing with the environment on one’s own, as in finding, storing and cooking food, and making stone tools. Another 30 percent came from cooperating to deal with the environment, such as banding together to hunt. The final 10 percent from competition between groups of people.\u003c/p>\n\u003cp>While the study didn’t specifically look at the impact of cultural tasks, it gave evidence of a substantial influence from them, too, Gonzalez-Forero said. He plans to assess cultural factors in the future.\u003c/p>\n\u003cp>In any case, the results are not intended to be the final word, but rather an encouragement for other researchers to use computer simulation for studying the question of brain size. Much exciting work remains to be done, he said.\u003c/p>\n\u003cp>Experts unconnected to the study were skeptical of the conclusions.\u003c/p>\n\u003cp>Dean Falk, a brain-evolution expert at Florida State University, said the work doesn’t assess the longstanding hypothesis that the development of language may have driven expansion of the brain. Gonzalez-Forero said the language idea is to some extent part of the cultural factors that remain to be addressed.\u003c/p>\n\u003cp>Falk also said the simulation strategy may have overemphasized the role of energy demands in influencing brain growth.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>Robin Dunbar, a professor of evolutionary psychology at Oxford University, and paleoanthropologist Richard Potts of the Smithsonian Institution said they didn’t think the simulation adequately mimicked the lives our ancestors lived.\u003c/p>\n\n\u003c/div>\u003c/p>",
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"disqusTitle": "New Snail Research Could Shake Up Our Understanding of Memory",
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"content": "\u003cp>UCLA neuroscientists reported Monday that they have transferred a memory from one animal to another via injections of RNA, a startling result that challenges the widely held view of where and how memories are stored in the brain. [contextly_sidebar id=\"li8sZlyeZueTl33XPD1EtLOaLArCiTWy\"]\u003c/p>\n\u003cp>The finding from the lab of David Glanzman hints at the potential for new RNA-based treatments to one day restore lost memories and, if correct, could shake up the field of memory and learning.\u003c/p>\n\u003cp>“It’s pretty shocking,” said Dr. Todd Sacktor, a neurologist and memory researcher at SUNY Downstate Medical Center in Brooklyn, N.Y. “The big picture is we’re working out the basic alphabet of how memories are stored for the first time.” He was not involved in the research, which was \u003ca href=\"http://www.eneuro.org/content/early/2018/05/14/ENEURO.0038-18.2018\" target=\"_blank\" rel=\"noopener\">published in eNeuro\u003c/a>, the \u003ca href=\"http://www.eneuro.org/\" target=\"_blank\" rel=\"noopener\">online journal\u003c/a> of the Society for Neuroscience.\u003c/p>\n\u003cp>Many scientists are expected to view the research more cautiously. The work is in snails, animals that have proven a powerful model organism for neuroscience but whose simple brains work far differently than those of humans. The experiments will need to be replicated, including in animals with more complex brains. And the results fly in the face of a massive amount of evidence supporting the deeply entrenched idea that memories are stored through changes in the strength of connections, or synapses, between neurons.\u003c/p>\n\u003cp>“If he’s right, this would be absolutely earth-shattering,” said Tomás Ryan, an assistant professor at Trinity College Dublin, whose lab hunts for engrams, or the physical traces of memory. “But I don’t think it’s right.”\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>Glanzman knows his unceremonial demotion of the synapse is not going to go over well in the field. “I expect a lot of astonishment and skepticism,” he said. “I don’t expect people are going to have a parade for me at the next Society for Neuroscience meeting.”\u003c/p>\n\u003cp>Even his own colleagues were dubious. “It took me a long time to convince the people in my lab to do the experiment,” he said. “They thought it was nuts.”\u003c/p>\n\u003cp>Glanzman’s experiments — funded by the National Institutes of Health and the National Science Foundation — involved giving mild electrical shocks to the marine snail Aplysia californica. Shocked snails learn to withdraw their delicate siphons and gills for nearly a minute as a defense when they subsequently receive a weak touch; snails that have not been shocked withdraw only briefly.[contextly_sidebar id=\"hBHp5EEY5u6iaZayEtFqvfhgpnNXWkKq\"]\u003c/p>\n\u003cp>The researchers extracted RNA from the nervous systems of snails that had been shocked and injected the material into unshocked snails. RNA’s primary role is to serve as a messenger inside cells, carrying protein-making instructions from its cousin DNA. But when this RNA was injected, these naive snails withdrew their siphons for extended periods of time after a soft touch. Control snails that received injections of RNA from snails that had not received shocks did not withdraw their siphons for as long.\u003c/p>\n\u003cp>“It’s as if we transferred a memory,” Glanzman said.\u003c/p>\n\u003cp>Glanzman’s group went further, showing that Aplysia sensory neurons in Petri dishes were more excitable, as they tend to be after being shocked, if they were exposed to RNA from shocked snails. Exposure to RNA from snails that had never been shocked did not cause the cells to become more excitable.\u003c/p>\n\u003cdiv>\n\u003cdiv>\n\u003cdiv>\n\u003cp>The results, said Glanzman, suggest that memories may be stored within the nucleus of neurons, where RNA is synthesized and can act on DNA to turn genes on and off. He said he thought memory storage involved these epigenetic changes — changes in the activity of genes and not in the DNA sequences that make up those genes — that are mediated by RNA.\u003c/p>\n\u003c/div>\n\u003c/div>\n\u003c/div>\n\u003cp>This view challenges the widely held notion that memories are stored by enhancing synaptic connections between neurons. Rather, Glanzman sees synaptic changes that occur during memory formation as flowing from the information that the RNA is carrying.\u003c/p>\n\u003cp>“This idea is radical and definitely challenges the field,” said Li-Huei Tsai, a neuroscientist who directs the Picower Institute for Learning and Memory at the Massachusetts Institute of Technology. Tsai, who recently co-authored a \u003ca href=\"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4874022/\" target=\"_blank\" rel=\"noopener\">major review on memory formation\u003c/a>, called Glanzman’s study “impressive and interesting” and said a number of studies support the notion that epigenetic mechanisms play some role in memory formation, which is likely a complex and multifaceted process. But she said she strongly disagreed with Glanzman’s notion that synaptic connections do not play a key role in memory storage.\u003c/p>\n\u003cp>Trinity College’s Ryan, like Glanzman, stands with a minority of neuroscientists — some call them rebels — who \u003ca href=\"https://www.ncbi.nlm.nih.gov/pubmed/28548457\" target=\"_blank\" rel=\"noopener\">question the idea\u003c/a> that memory is stored through synaptic strength. In 2015, Ryan was lead author of a Science paper with MIT Nobelist Susumu Tonegawa that showed \u003ca href=\"https://www.ncbi.nlm.nih.gov/pubmed/26023136\" target=\"_blank\" rel=\"noopener\">memories could be retrieved\u003c/a> even after synapse strengthening was blocked. Ryan said he is pursuing the idea that memories are stored through ensembles of neurons bound together by new synaptic connections, not by strengthening of existing connections.[contextly_sidebar id=\"U81WeRFYsnSgGUp9A3OIFy9mt8TlGFLL\"]\u003c/p>\n\u003cp>Ryan knows Glanzman and trusts his work. He said he believes the data in the new paper. But he doesn’t think the behavior of the snails, or the cells, proves that RNA is transferring memories. He said he doesn’t understand how RNA, which works on a time scale of minutes to hours, could be causing memory recall that is almost instantaneous, or how RNA could connect numerous parts of the brain, like the auditory and visual systems, that are involved in more complex memories.\u003c/p>\n\u003cp>But Glanzman said he is convinced RNA is playing a role that eclipses the synapse. In 2014, his lab showed that \u003ca href=\"https://elifesciences.org/articles/03896\" target=\"_blank\" rel=\"noopener\">memories of shocks that had been lost\u003c/a> in snails due to a series of experimental procedures could be recovered — but the synapse patterns that were lost with the memory reformed in random ways when the memories were recovered, suggesting memories were not stored there. Glanzman’s lab and others have also shown that long-term memory formation can be blocked by preventing epigenetic changes, even when synapse formation or strengthening is not altered.\u003c/p>\n\u003cp>“Synapses can come and go, but the memory can still be there,” he said, saying he sees synapses as merely the “reflection of knowledge held in the nucleus.”\u003c/p>\n\u003cp>Glanzman has studied memory for more than three decades. He did postdoctoral work with none other than Eric Kandel — the neuroscientist who shared the 2000 Nobel prize for research on Aplysia, probing the role of the synapse in memory — and he said he has spent most of his career believing that synaptic change was the key to memory storage.\u003c/p>\n\u003cp>But he said a series of findings from other labs and his own in recent years have led him to start questioning the synaptic dogma. He calls himself “a recovering synaptologist.”\u003c/p>\n\u003cp>The skepticism over Glanzman’s research may be in part because the work harkens back to an unnerving episode in science involving an unconventional psychologist, James V. McConnell, who spent years at the University of Michigan attempting to prove that something outside the brain — a factor he called “memory RNA” — could transfer memories. In the ’50s and ’60s, McConnell trained flatworms and then fed the bodies of trained worms to untrained worms. The untrained worms then appeared to exhibit the behavior of the trained worms they’d cannibalized, suggesting that memories were somehow transferred. He also showed that trained worms that were beheaded could remember their training after they grew new heads.[contextly_sidebar id=\"F5HcwGf5fX0A2ZqbT4U7VYdgDDaJshog\"]\u003c/p>\n\u003cp>Though the work was replicated by some other labs, McConnell’s work was largely ridiculed and is often described as a cautionary tale because so much time and money was spent by other labs trying, often unsuccessfully, to replicate the work. (McConnell died in 1990, five years after he’d been a target of the Unabomber Theodore Kaczynski.)\u003c/p>\n\u003cp>Recently, developmental biologist Michael Levin at Tufts has \u003ca href=\"https://www.theverge.com/2015/3/18/8225321/memory-research-flatworm-cannibalism-james-mcconnell-michael-levin\" target=\"_blank\" rel=\"noopener\">replicated McConnell’s experiments\u003c/a> on headless worms under more controlled settings and thinks McConnell may have indeed been correct.\u003c/p>\n\u003cp>Glanzman said one of McConnell’s students, Al Jacobson, demonstrated the transfer of memories between flatworms via RNA injections, coincidentally while an assistant professor at UCLA. The work was \u003ca href=\"http://www.nature.com/articles/209599a0\" target=\"_blank\" rel=\"noopener\">published in Nature in 1966\u003c/a> but Jacobsen never received tenure, perhaps because of doubts about his findings. The experiment was, however, \u003ca href=\"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC224185/\" target=\"_blank\" rel=\"noopener\">replicated in rats\u003c/a> shortly afterward.\u003c/p>\n\u003cp>Glanzman learned about McConnell’s work — and his satirical journal “Worm Runner’s Digest” — while he was a psychology undergraduate at Indiana University but never took the results seriously. Now, while he’s still not convinced McConnell was exactly right about being able to transfer memories, he does think both McConnell and Jacobson were onto something.\u003c/p>\n\u003cp>Working in the memory field can be tough for those who challenge the status quo. SUNY’s Sacktor, for example, has \u003ca href=\"https://www.statnews.com/2016/06/23/memory-research-neuroscience/\">spent more than 25 years\u003c/a> — despite the skepticism, rejection, and outright derision of fellow scientists — chasing down a single molecule, PKMzeta, that he believes is critical to the formation of long-term memories and may be connected to the RNA mechanisms that Glanzman has uncovered.\u003c/p>\n\u003cp>The stakes in the field are high because memory is so key to our sense of self and many scientists feel understanding the workings of memory is something that should have been figured out by now. “It’s the last of the great 20th-century questions in biology,” Sacktor said. “Some aspect has made it difficult for neuroscientists to figure out.”\u003c/p>\n\u003cp>The difficulty may be due in part to the overwhelming focus on synaptic strength. Some 12,000 papers have been published on synaptic strength without providing a good explanation for how memories are stored, Ryan noted, adding that he applauds Glanzman for opening up a new path, radical as it is, to explore.\u003c/p>\n\u003cp>“The reality is we know so little about memory,” Ryan said. “I’m excited about any new vistas and avenues.”\u003c/p>\n\u003cp>[ad floatright]\u003c/p>\n\u003cp>\u003cem>This \u003ca href=\"https://www.statnews.com/2018/05/14/memory-transfer-between-snails-challenges-standard-theory/\" target=\"_blank\" rel=\"noopener\">story\u003c/a> was originally published by STAT, an online publication of Boston Globe Media that covers health, medicine, and scientific discovery.\u003c/em>\u003c/p>\n\n",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp>UCLA neuroscientists reported Monday that they have transferred a memory from one animal to another via injections of RNA, a startling result that challenges the widely held view of where and how memories are stored in the brain. \u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>The finding from the lab of David Glanzman hints at the potential for new RNA-based treatments to one day restore lost memories and, if correct, could shake up the field of memory and learning.\u003c/p>\n\u003cp>“It’s pretty shocking,” said Dr. Todd Sacktor, a neurologist and memory researcher at SUNY Downstate Medical Center in Brooklyn, N.Y. “The big picture is we’re working out the basic alphabet of how memories are stored for the first time.” He was not involved in the research, which was \u003ca href=\"http://www.eneuro.org/content/early/2018/05/14/ENEURO.0038-18.2018\" target=\"_blank\" rel=\"noopener\">published in eNeuro\u003c/a>, the \u003ca href=\"http://www.eneuro.org/\" target=\"_blank\" rel=\"noopener\">online journal\u003c/a> of the Society for Neuroscience.\u003c/p>\n\u003cp>Many scientists are expected to view the research more cautiously. The work is in snails, animals that have proven a powerful model organism for neuroscience but whose simple brains work far differently than those of humans. The experiments will need to be replicated, including in animals with more complex brains. And the results fly in the face of a massive amount of evidence supporting the deeply entrenched idea that memories are stored through changes in the strength of connections, or synapses, between neurons.\u003c/p>\n\u003cp>“If he’s right, this would be absolutely earth-shattering,” said Tomás Ryan, an assistant professor at Trinity College Dublin, whose lab hunts for engrams, or the physical traces of memory. “But I don’t think it’s right.”\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>Glanzman knows his unceremonial demotion of the synapse is not going to go over well in the field. “I expect a lot of astonishment and skepticism,” he said. “I don’t expect people are going to have a parade for me at the next Society for Neuroscience meeting.”\u003c/p>\n\u003cp>Even his own colleagues were dubious. “It took me a long time to convince the people in my lab to do the experiment,” he said. “They thought it was nuts.”\u003c/p>\n\u003cp>Glanzman’s experiments — funded by the National Institutes of Health and the National Science Foundation — involved giving mild electrical shocks to the marine snail Aplysia californica. Shocked snails learn to withdraw their delicate siphons and gills for nearly a minute as a defense when they subsequently receive a weak touch; snails that have not been shocked withdraw only briefly.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>The researchers extracted RNA from the nervous systems of snails that had been shocked and injected the material into unshocked snails. RNA’s primary role is to serve as a messenger inside cells, carrying protein-making instructions from its cousin DNA. But when this RNA was injected, these naive snails withdrew their siphons for extended periods of time after a soft touch. Control snails that received injections of RNA from snails that had not received shocks did not withdraw their siphons for as long.\u003c/p>\n\u003cp>“It’s as if we transferred a memory,” Glanzman said.\u003c/p>\n\u003cp>Glanzman’s group went further, showing that Aplysia sensory neurons in Petri dishes were more excitable, as they tend to be after being shocked, if they were exposed to RNA from shocked snails. Exposure to RNA from snails that had never been shocked did not cause the cells to become more excitable.\u003c/p>\n\u003cdiv>\n\u003cdiv>\n\u003cdiv>\n\u003cp>The results, said Glanzman, suggest that memories may be stored within the nucleus of neurons, where RNA is synthesized and can act on DNA to turn genes on and off. He said he thought memory storage involved these epigenetic changes — changes in the activity of genes and not in the DNA sequences that make up those genes — that are mediated by RNA.\u003c/p>\n\u003c/div>\n\u003c/div>\n\u003c/div>\n\u003cp>This view challenges the widely held notion that memories are stored by enhancing synaptic connections between neurons. Rather, Glanzman sees synaptic changes that occur during memory formation as flowing from the information that the RNA is carrying.\u003c/p>\n\u003cp>“This idea is radical and definitely challenges the field,” said Li-Huei Tsai, a neuroscientist who directs the Picower Institute for Learning and Memory at the Massachusetts Institute of Technology. Tsai, who recently co-authored a \u003ca href=\"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4874022/\" target=\"_blank\" rel=\"noopener\">major review on memory formation\u003c/a>, called Glanzman’s study “impressive and interesting” and said a number of studies support the notion that epigenetic mechanisms play some role in memory formation, which is likely a complex and multifaceted process. But she said she strongly disagreed with Glanzman’s notion that synaptic connections do not play a key role in memory storage.\u003c/p>\n\u003cp>Trinity College’s Ryan, like Glanzman, stands with a minority of neuroscientists — some call them rebels — who \u003ca href=\"https://www.ncbi.nlm.nih.gov/pubmed/28548457\" target=\"_blank\" rel=\"noopener\">question the idea\u003c/a> that memory is stored through synaptic strength. In 2015, Ryan was lead author of a Science paper with MIT Nobelist Susumu Tonegawa that showed \u003ca href=\"https://www.ncbi.nlm.nih.gov/pubmed/26023136\" target=\"_blank\" rel=\"noopener\">memories could be retrieved\u003c/a> even after synapse strengthening was blocked. Ryan said he is pursuing the idea that memories are stored through ensembles of neurons bound together by new synaptic connections, not by strengthening of existing connections.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>Ryan knows Glanzman and trusts his work. He said he believes the data in the new paper. But he doesn’t think the behavior of the snails, or the cells, proves that RNA is transferring memories. He said he doesn’t understand how RNA, which works on a time scale of minutes to hours, could be causing memory recall that is almost instantaneous, or how RNA could connect numerous parts of the brain, like the auditory and visual systems, that are involved in more complex memories.\u003c/p>\n\u003cp>But Glanzman said he is convinced RNA is playing a role that eclipses the synapse. In 2014, his lab showed that \u003ca href=\"https://elifesciences.org/articles/03896\" target=\"_blank\" rel=\"noopener\">memories of shocks that had been lost\u003c/a> in snails due to a series of experimental procedures could be recovered — but the synapse patterns that were lost with the memory reformed in random ways when the memories were recovered, suggesting memories were not stored there. Glanzman’s lab and others have also shown that long-term memory formation can be blocked by preventing epigenetic changes, even when synapse formation or strengthening is not altered.\u003c/p>\n\u003cp>“Synapses can come and go, but the memory can still be there,” he said, saying he sees synapses as merely the “reflection of knowledge held in the nucleus.”\u003c/p>\n\u003cp>Glanzman has studied memory for more than three decades. He did postdoctoral work with none other than Eric Kandel — the neuroscientist who shared the 2000 Nobel prize for research on Aplysia, probing the role of the synapse in memory — and he said he has spent most of his career believing that synaptic change was the key to memory storage.\u003c/p>\n\u003cp>But he said a series of findings from other labs and his own in recent years have led him to start questioning the synaptic dogma. He calls himself “a recovering synaptologist.”\u003c/p>\n\u003cp>The skepticism over Glanzman’s research may be in part because the work harkens back to an unnerving episode in science involving an unconventional psychologist, James V. McConnell, who spent years at the University of Michigan attempting to prove that something outside the brain — a factor he called “memory RNA” — could transfer memories. In the ’50s and ’60s, McConnell trained flatworms and then fed the bodies of trained worms to untrained worms. The untrained worms then appeared to exhibit the behavior of the trained worms they’d cannibalized, suggesting that memories were somehow transferred. He also showed that trained worms that were beheaded could remember their training after they grew new heads.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>Though the work was replicated by some other labs, McConnell’s work was largely ridiculed and is often described as a cautionary tale because so much time and money was spent by other labs trying, often unsuccessfully, to replicate the work. (McConnell died in 1990, five years after he’d been a target of the Unabomber Theodore Kaczynski.)\u003c/p>\n\u003cp>Recently, developmental biologist Michael Levin at Tufts has \u003ca href=\"https://www.theverge.com/2015/3/18/8225321/memory-research-flatworm-cannibalism-james-mcconnell-michael-levin\" target=\"_blank\" rel=\"noopener\">replicated McConnell’s experiments\u003c/a> on headless worms under more controlled settings and thinks McConnell may have indeed been correct.\u003c/p>\n\u003cp>Glanzman said one of McConnell’s students, Al Jacobson, demonstrated the transfer of memories between flatworms via RNA injections, coincidentally while an assistant professor at UCLA. The work was \u003ca href=\"http://www.nature.com/articles/209599a0\" target=\"_blank\" rel=\"noopener\">published in Nature in 1966\u003c/a> but Jacobsen never received tenure, perhaps because of doubts about his findings. The experiment was, however, \u003ca href=\"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC224185/\" target=\"_blank\" rel=\"noopener\">replicated in rats\u003c/a> shortly afterward.\u003c/p>\n\u003cp>Glanzman learned about McConnell’s work — and his satirical journal “Worm Runner’s Digest” — while he was a psychology undergraduate at Indiana University but never took the results seriously. Now, while he’s still not convinced McConnell was exactly right about being able to transfer memories, he does think both McConnell and Jacobson were onto something.\u003c/p>\n\u003cp>Working in the memory field can be tough for those who challenge the status quo. SUNY’s Sacktor, for example, has \u003ca href=\"https://www.statnews.com/2016/06/23/memory-research-neuroscience/\">spent more than 25 years\u003c/a> — despite the skepticism, rejection, and outright derision of fellow scientists — chasing down a single molecule, PKMzeta, that he believes is critical to the formation of long-term memories and may be connected to the RNA mechanisms that Glanzman has uncovered.\u003c/p>\n\u003cp>The stakes in the field are high because memory is so key to our sense of self and many scientists feel understanding the workings of memory is something that should have been figured out by now. “It’s the last of the great 20th-century questions in biology,” Sacktor said. “Some aspect has made it difficult for neuroscientists to figure out.”\u003c/p>\n\u003cp>The difficulty may be due in part to the overwhelming focus on synaptic strength. Some 12,000 papers have been published on synaptic strength without providing a good explanation for how memories are stored, Ryan noted, adding that he applauds Glanzman for opening up a new path, radical as it is, to explore.\u003c/p>\n\u003cp>“The reality is we know so little about memory,” Ryan said. “I’m excited about any new vistas and avenues.”\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cp>Author Michael Pollan had always been curious about psychoactive plants, but his interest skyrocketed when he heard about a \u003ca href=\"https://www.npr.org/sections/health-shots/2016/12/03/504136736/how-a-psychedelic-drug-helps-cancer-patients-overcome-anxiety\" target=\"_blank\" rel=\"noopener\">research study\u003c/a> in which people with terminal cancer were given a psychedelic called \u003ca href=\"https://www.npr.org/tags/140958387/psilocybin\" target=\"_blank\" rel=\"noopener\">psilocybin \u003c/a>— the active ingredient in \"magic mushrooms\" — to help them deal with their distress.\u003c/p>\n\u003cp>\"This seemed like such a crazy idea that I began looking into it,\" Pollan says. \"Why should a drug from a mushroom help people deal with their mortality?\"\u003c/p>\n\u003cp>Pollan, whose previous books include \u003ca href=\"https://www.npr.org/books/titles/137946336/the-omnivores-dilemma-a-natural-history-of-four-meals\" target=\"_blank\" rel=\"noopener\">The Omnivore's Dilemma\u003c/a> and \u003ca href=\"https://www.npr.org/templates/story/story.php?storyId=17725932\" target=\"_blank\" rel=\"noopener\">In Defense Of Food\u003c/a>, started researching different experimental therapeutic uses of psychedelics, and found that the drugs were being used to treat depression, addiction and the fear of death.\u003c/p>\n\u003cp>Then he decided to go one step further: A self-described \"reluctant psychonaut,\" Pollan enlisted guides to help him experiment with LSD, psilocybin and 5-MeO-DMT, a substance in the venom of the Sonoran Desert toad.\u003c/p>\n\u003cp>Each of Pollan's experiences with psychedelics was preceded by worry and self-doubt. But, he says, \"I realized later that was my ego trying to convince me not to do this thing that was going to challenge my ego.\"\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>Pollan's new book, \u003cem>How to Change Your Mind: What the New Science of Psychedelics Teaches Us About Consciousness, Dying, Addiction, Depression, and Transcendence\u003c/em>, recounts his experiences with the drugs and also examines the history of psychedelics as well as their possible therapeutic uses.\u003c/p>\n\u003chr>\n\u003cp>\u003cstrong>Interview Highlights\u003c/strong>\u003c/p>\n\u003cp>\u003cstrong>On how the psychedelic psilocybin is administered in therapy for depression \u003c/strong>\u003c/p>\n\u003cp>The way [psilocybin is] being used is in a very controlled or guided setting. ... They don't just give you a pill and send you home; you're in a room. You're with two guides, one male, one female. You're lying down on a comfortable couch. You're wearing headphones listening to a really carefully curated playlist of music — instrumental compositions for the most part — and you're wearing eyeshades, all of which is to encourage a very inward journey.\u003c/p>\n\u003cp>Someone is kind of looking out for you, and they prepare you very carefully in advance. They give you a set of \"flight instructions,\" as they call them, which is what to do if you get really scared or you're beginning to have a bad trip. If you see a monster, for example, don't try to run away. Walk right up to it, plant your feet and say, \"What do you have to teach me? What are you doing in my mind?\" And if you do that, according to the flight instructions, your fear will morph into something much more positive very quickly.\u003c/p>\n\u003cp>\u003cstrong>On how psychedelics can help change the stories we tell about ourselves \u003c/strong>\u003c/p>\n\u003cp>The drugs foster new perspectives on old problems. One of the things our mind does is tell stories about ourselves. If you're depressed, you're being told a story perhaps that you're worthless, that no one could possibly love you, you're not worthy of love, that life will not get better. And these stories — which are enforced by our egos really — trap us in these ruminative loops that are very hard to get out of. They're very destructive patterns of thought.\u003c/p>\n\u003cp>What the drugs appear to do is disable for a period of time the part of the brain where the self talks to itself. It's called the \u003ca href=\"https://www.npr.org/tags/482443062/default-mode-network\" target=\"_blank\" rel=\"noopener\">default mode network\u003c/a>, and it's a group of structures that connect parts of the cortex — the evolutionarily most recent part of the brain — to deeper levels where emotion and memory reside. And it's a very important hub in the brain and lots of important things happen there: self-reflection and rumination, time travel. It's where we go to think about the future or the past, and theory of mind, the ability to imagine the mental states of other beings and, I think, most importantly, the autobiographical self. It's the part of the brain, it appears, where we incorporate things that happen to us, new information, with a sense of who we are, who we were and who we want to be. And that's where these stories get generated. And these stories can be really destructive, they trap us. ...\u003c/p>\n\u003cp>This network is downregulated [with psychedelics], it sort of goes offline for a period of time. And that's why you experience this dissolution of self or ego, which can be a terrifying or liberating thing, depending on your mindset. This is what allows people, I think, to have those new perspectives on themselves, to realize that they needn't be trapped in those stories and they might actually be able to write some new stories about themselves. That's what's liberating, I think, about the experience when it works.\u003c/p>\n\u003cp>\u003cstrong>On how psychedelics can help dying people face their deaths \u003c/strong>\u003c/p>\n\u003cp>\u003ca href=\"https://www.npr.org/tags/125941537/prozac\">Prozac\u003c/a> doesn't help when you're confronting your mortality. But here we have something that occasions an experience in people — a mystical experience — that somehow makes it easier to let go. And I think some of it has to do with the fact that you do experience the \"extinction\" of yourself and it's kind of a rehearsal for death. And I think that may be part of what helps people, that they expand their sense of what is your self-interest and your self-interest is something larger than what is contained by your skin. And when you have that recognition, I think dying becomes a little easier. ...\u003c/p>\n\u003cp>There's no way to prove this, obviously, and it's a question that really troubled me as an old-fashioned materialist skeptical journalist. It's like, \"What if these drugs are inducing an illusion in people?\" I got a variety of answers to that question from the researchers. One was, \"Who cares if it helps them?\" And I can see the point of that. The other was, \"Hey, this is beyond my pay grade; none of us know what happens after we die.\" And others say, \"Well, this is an open frontier.\" ...\u003c/p>\n\u003cp>The experiences that people have are very real to them — they're psychological facts. And one of the really interesting qualities of psychedelic experience is that the insights you have on them have a durability ... This isn't just an opinion, this is revealed truth, so the confidence people have is hard to shake, actually.\u003c/p>\n\u003cp>\u003cstrong>On a \u003c/strong>\u003ca href=\"https://www.hopkinsmedicine.org/news/stories/mushrooms_quit_smoking.html\" target=\"_blank\" rel=\"noopener\">\u003cstrong>Johns Hopkins study\u003c/strong>\u003c/a>\u003cstrong> on the use of psilocybin to help people quit smoking \u003c/strong>\u003c/p>\n\u003cp>Smoking is a very hard addiction to break. It's one of the hardest addictions to break. [I wanted to understand] how, after a single psilocybin trip, they could decide \"I'm never going to smoke again\" based on the perspective they had achieved. And they would say things like, \"Well, I had this amazing experience. I died three times. I sprouted wings. I flew through European histories. I beheld all these wonders. I saw my body on a funeral pyre on the Ganges. And I realized, the universe is so amazing and there's so much to do in it that killing myself seemed really stupid.\" And that was the insight. Yes, killing yourself is really stupid — but it had an authority it had never had. And that, I think, is the gift of these psychedelics.\u003c/p>\n\u003cp>\u003cstrong>On his own experience tripping on mushrooms \u003c/strong>\u003c/p>\n\u003cp>I had an experience that was by turns frightening and ecstatic and weird. ... I found myself in this place where I could no longer control my perceptions at all, and I felt my sense of self scatter to the wind — almost as if a pile of post-its had been released to the wind — but I was fine with it. I didn't feel any desire to pile the papers back together into my customary self ...\u003c/p>\n\u003cp>Then I looked out and saw myself spread over the landscape like a coat of paint or butter. I was outside myself, beside myself, literally, and the consciousness that beheld this ... was not my normal consciousness, it was completely unperturbed. It was dispassionate. It was content, as I watched myself dissolve over the landscape.\u003c/p>\n\u003cp>What I brought back from that experience was that I'm not identical to my ego, that there is another ground on which to plant our feet and that our ego is kind of this character that is chattering neurotically in our minds. And it's good for lots of things. I mean, the ego got the book written, but it also can be very harsh, and it's liberating to have some distance on it. And that was a great gift, I think.\u003c/p>\n\u003cp>[ad floatright]\u003c/p>\n\u003cp>\u003cem>Sam Briger and Seth Kelley produced and edited this interview for broadcast. Bridget Bentz, Molly Seavy-Nesper and Scott Hensley adapted it for the Web.\u003c/em>\u003c/p>\n\u003cdiv class=\"fullattribution\">Copyright 2018 Fresh Air. 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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp>Author Michael Pollan had always been curious about psychoactive plants, but his interest skyrocketed when he heard about a \u003ca href=\"https://www.npr.org/sections/health-shots/2016/12/03/504136736/how-a-psychedelic-drug-helps-cancer-patients-overcome-anxiety\" target=\"_blank\" rel=\"noopener\">research study\u003c/a> in which people with terminal cancer were given a psychedelic called \u003ca href=\"https://www.npr.org/tags/140958387/psilocybin\" target=\"_blank\" rel=\"noopener\">psilocybin \u003c/a>— the active ingredient in \"magic mushrooms\" — to help them deal with their distress.\u003c/p>\n\u003cp>\"This seemed like such a crazy idea that I began looking into it,\" Pollan says. \"Why should a drug from a mushroom help people deal with their mortality?\"\u003c/p>\n\u003cp>Pollan, whose previous books include \u003ca href=\"https://www.npr.org/books/titles/137946336/the-omnivores-dilemma-a-natural-history-of-four-meals\" target=\"_blank\" rel=\"noopener\">The Omnivore's Dilemma\u003c/a> and \u003ca href=\"https://www.npr.org/templates/story/story.php?storyId=17725932\" target=\"_blank\" rel=\"noopener\">In Defense Of Food\u003c/a>, started researching different experimental therapeutic uses of psychedelics, and found that the drugs were being used to treat depression, addiction and the fear of death.\u003c/p>\n\u003cp>Then he decided to go one step further: A self-described \"reluctant psychonaut,\" Pollan enlisted guides to help him experiment with LSD, psilocybin and 5-MeO-DMT, a substance in the venom of the Sonoran Desert toad.\u003c/p>\n\u003cp>Each of Pollan's experiences with psychedelics was preceded by worry and self-doubt. But, he says, \"I realized later that was my ego trying to convince me not to do this thing that was going to challenge my ego.\"\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>Pollan's new book, \u003cem>How to Change Your Mind: What the New Science of Psychedelics Teaches Us About Consciousness, Dying, Addiction, Depression, and Transcendence\u003c/em>, recounts his experiences with the drugs and also examines the history of psychedelics as well as their possible therapeutic uses.\u003c/p>\n\u003chr>\n\u003cp>\u003cstrong>Interview Highlights\u003c/strong>\u003c/p>\n\u003cp>\u003cstrong>On how the psychedelic psilocybin is administered in therapy for depression \u003c/strong>\u003c/p>\n\u003cp>The way [psilocybin is] being used is in a very controlled or guided setting. ... They don't just give you a pill and send you home; you're in a room. You're with two guides, one male, one female. You're lying down on a comfortable couch. You're wearing headphones listening to a really carefully curated playlist of music — instrumental compositions for the most part — and you're wearing eyeshades, all of which is to encourage a very inward journey.\u003c/p>\n\u003cp>Someone is kind of looking out for you, and they prepare you very carefully in advance. They give you a set of \"flight instructions,\" as they call them, which is what to do if you get really scared or you're beginning to have a bad trip. If you see a monster, for example, don't try to run away. Walk right up to it, plant your feet and say, \"What do you have to teach me? What are you doing in my mind?\" And if you do that, according to the flight instructions, your fear will morph into something much more positive very quickly.\u003c/p>\n\u003cp>\u003cstrong>On how psychedelics can help change the stories we tell about ourselves \u003c/strong>\u003c/p>\n\u003cp>The drugs foster new perspectives on old problems. One of the things our mind does is tell stories about ourselves. If you're depressed, you're being told a story perhaps that you're worthless, that no one could possibly love you, you're not worthy of love, that life will not get better. And these stories — which are enforced by our egos really — trap us in these ruminative loops that are very hard to get out of. They're very destructive patterns of thought.\u003c/p>\n\u003cp>What the drugs appear to do is disable for a period of time the part of the brain where the self talks to itself. It's called the \u003ca href=\"https://www.npr.org/tags/482443062/default-mode-network\" target=\"_blank\" rel=\"noopener\">default mode network\u003c/a>, and it's a group of structures that connect parts of the cortex — the evolutionarily most recent part of the brain — to deeper levels where emotion and memory reside. And it's a very important hub in the brain and lots of important things happen there: self-reflection and rumination, time travel. It's where we go to think about the future or the past, and theory of mind, the ability to imagine the mental states of other beings and, I think, most importantly, the autobiographical self. It's the part of the brain, it appears, where we incorporate things that happen to us, new information, with a sense of who we are, who we were and who we want to be. And that's where these stories get generated. And these stories can be really destructive, they trap us. ...\u003c/p>\n\u003cp>This network is downregulated [with psychedelics], it sort of goes offline for a period of time. And that's why you experience this dissolution of self or ego, which can be a terrifying or liberating thing, depending on your mindset. This is what allows people, I think, to have those new perspectives on themselves, to realize that they needn't be trapped in those stories and they might actually be able to write some new stories about themselves. That's what's liberating, I think, about the experience when it works.\u003c/p>\n\u003cp>\u003cstrong>On how psychedelics can help dying people face their deaths \u003c/strong>\u003c/p>\n\u003cp>\u003ca href=\"https://www.npr.org/tags/125941537/prozac\">Prozac\u003c/a> doesn't help when you're confronting your mortality. But here we have something that occasions an experience in people — a mystical experience — that somehow makes it easier to let go. And I think some of it has to do with the fact that you do experience the \"extinction\" of yourself and it's kind of a rehearsal for death. And I think that may be part of what helps people, that they expand their sense of what is your self-interest and your self-interest is something larger than what is contained by your skin. And when you have that recognition, I think dying becomes a little easier. ...\u003c/p>\n\u003cp>There's no way to prove this, obviously, and it's a question that really troubled me as an old-fashioned materialist skeptical journalist. It's like, \"What if these drugs are inducing an illusion in people?\" I got a variety of answers to that question from the researchers. One was, \"Who cares if it helps them?\" And I can see the point of that. The other was, \"Hey, this is beyond my pay grade; none of us know what happens after we die.\" And others say, \"Well, this is an open frontier.\" ...\u003c/p>\n\u003cp>The experiences that people have are very real to them — they're psychological facts. And one of the really interesting qualities of psychedelic experience is that the insights you have on them have a durability ... This isn't just an opinion, this is revealed truth, so the confidence people have is hard to shake, actually.\u003c/p>\n\u003cp>\u003cstrong>On a \u003c/strong>\u003ca href=\"https://www.hopkinsmedicine.org/news/stories/mushrooms_quit_smoking.html\" target=\"_blank\" rel=\"noopener\">\u003cstrong>Johns Hopkins study\u003c/strong>\u003c/a>\u003cstrong> on the use of psilocybin to help people quit smoking \u003c/strong>\u003c/p>\n\u003cp>Smoking is a very hard addiction to break. It's one of the hardest addictions to break. [I wanted to understand] how, after a single psilocybin trip, they could decide \"I'm never going to smoke again\" based on the perspective they had achieved. And they would say things like, \"Well, I had this amazing experience. I died three times. I sprouted wings. I flew through European histories. I beheld all these wonders. I saw my body on a funeral pyre on the Ganges. And I realized, the universe is so amazing and there's so much to do in it that killing myself seemed really stupid.\" And that was the insight. Yes, killing yourself is really stupid — but it had an authority it had never had. And that, I think, is the gift of these psychedelics.\u003c/p>\n\u003cp>\u003cstrong>On his own experience tripping on mushrooms \u003c/strong>\u003c/p>\n\u003cp>I had an experience that was by turns frightening and ecstatic and weird. ... I found myself in this place where I could no longer control my perceptions at all, and I felt my sense of self scatter to the wind — almost as if a pile of post-its had been released to the wind — but I was fine with it. I didn't feel any desire to pile the papers back together into my customary self ...\u003c/p>\n\u003cp>Then I looked out and saw myself spread over the landscape like a coat of paint or butter. I was outside myself, beside myself, literally, and the consciousness that beheld this ... was not my normal consciousness, it was completely unperturbed. It was dispassionate. It was content, as I watched myself dissolve over the landscape.\u003c/p>\n\u003cp>What I brought back from that experience was that I'm not identical to my ego, that there is another ground on which to plant our feet and that our ego is kind of this character that is chattering neurotically in our minds. And it's good for lots of things. I mean, the ego got the book written, but it also can be very harsh, and it's liberating to have some distance on it. And that was a great gift, I think.\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cp>A \u003ca href=\"https://leginfo.legislature.ca.gov/faces/billNavClient.xhtml?bill_id=201720180AB2108\" target=\"_blank\" rel=\"noopener\">bill\u003c/a> that would have made California the first state to ban youth tackle football was shelved at the end of April, following major \u003ca href=\"https://www.ocregister.com/2018/04/27/law-to-ban-tackle-football-for-california-youth-falls-short-of-committee/\" target=\"_blank\" rel=\"noopener\">pushback\u003c/a> from parents, coaches and football fans.[contextly_sidebar id=”3BKDehAqDVxXqjXF1gjQRNg4qvtqDn2e”]\u003c/p>\n\u003cp>Organizers collected more than 45,000 signatures opposing the ban, according to the \u003ca href=\"https://www.vcstar.com/story/news/2018/04/27/lawmakers-sideline-bill-ban-preteen-tackle-football/560916002/\" target=\"_blank\" rel=\"noopener\">Los Angeles Times\u003c/a>.\u003c/p>\n\u003cp>The \u003ca href=\"https://www.ocregister.com/2018/04/27/law-to-ban-tackle-football-for-california-youth-falls-short-of-committee/\" target=\"_blank\" rel=\"noopener\">Orange County Register\u003c/a> reported …\u003c/p>\n\u003cblockquote>\u003cp>(T)he backlash was swift and unyielding. Coaches and parents flooded phone lines, email boxes and talk radio, condemning the bills and questioning the science behind them. Within weeks, three of five bills were dead or left in legislative purgatory, crushed under the weight of youth football’s influence.\u003c/p>\u003c/blockquote>\n\u003caside class=\"pullquote alignright\">‘I have a problem with the idea that we’re going to take kids that we already know are vulnerable, and we are gonna hit them in the head a few hundred times a year.’ \u003ccite>Christopher Nowinski, co-founder of Boston University’s CTE Center\u003c/cite>\u003c/aside>\n\u003cp> \u003c/p>\n\u003cp>The \u003ca href=\"https://leginfo.legislature.ca.gov/faces/billNavClient.xhtml?bill_id=201720180AB2108\" target=\"_blank\" rel=\"noopener\">Safe Youth Football Act\u003c/a> would have barred children below age 12 from playing organized tackle football. The legislation was drafted in consultation with medical professionals who say the ban would protect youth from a degenerative brain disease known as \u003ca href=\"https://www.mayoclinic.org/diseases-conditions/chronic-traumatic-encephalopathy/symptoms-causes/syc-20370921\" target=\"_blank\" rel=\"noopener\">chronic traumatic encephalopathy\u003c/a>.\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>Those views were bolstered by a \u003ca href=\"https://www.bumc.bu.edu/busm/2018/04/30/youth-tackle-football-linked-to-earlier-onset-of-cognitive-and-emotional-symptoms-in-athletes-diagnosed-with-cte-after-death/\" target=\"_blank\" rel=\"noopener\">study \u003c/a>published April 30 that found playing youth tackle football may lead to earlier onset of cognitive, behavioral and emotional symptoms in later life.\u003c/p>\n\u003cp>Researchers at the Boston University School of Medicine conducted telephone interviews with the friends and relatives of 246 deceased football players and found that those who played tackle football before 12 experienced symptoms on average 13 years earlier than those who started playing at an older age.\u003c/p>\n\u003cp>Researchers took into account different styles of play and forms of protection.\u003c/p>\n\u003cp>“It took looking at 246 brains and seeing this in nearly 90 percent of them to say, ‘Hey, you know what, the research is becoming very clear, that you hit someone in the head too much, you’ll give them this disease,’ ” Christopher Nowinski, co-founder of Boston University’s \u003ca href=\"https://www.bu.edu/cte/\" target=\"_blank\" rel=\"noopener\">CTE Center\u003c/a>, said on \u003ca href=\"https://www.kqed.org/forum/2010101865041/after-california-ban-fails-controversy-persists-over-youth-tackle-football\" target=\"_blank\" rel=\"noopener\">KQED’s Forum program \u003c/a>last week.\u003c/p>\n\u003cp>Joe Rafter, the president of Southern Marin Youth Football who started a group to oppose the ban, disputes those findings and says the sport is “safer than its ever been.”[contextly_sidebar id=”mIxCIh40B3GwZE3hvjgDhmoIUuc7rwZy”]\u003c/p>\n\u003cp>He pointed to revised coaching methods and behavior modification techniques that he argues have made the sport safer.\u003c/p>\n\u003cp>“The game that was played 40 to 50 years ago is no longer played now,” he said. “The techniques that are taught right now in youth football across America are to take the head out of the tackle.”\u003c/p>\n\u003cp>\u003cstrong>Public Health Issue\u003c/strong>\u003c/p>\n\u003cp>But Nowinski, a former football player, says changes made in the sport are not enough to make it safe for youth. The problem is not the occasional major hit to the head, he said, but the repetitive tackling, hitting and blocking.\u003c/p>\n\u003cp>“This is the best data that we are going to have for decades, because we have taken the effort to actually build the world’s largest brain bank of former football players, and we just really struggle to find football players who don’t have CTE who’ve had long careers,” he said. “Even if we diagnose concussions, a concussion happens maybe once out of every couple thousand of times a child is hit in the head. CTE risk is related to those thousands of hits, not to that concussion.”\u003c/p>\n\u003cp>Nowinski says the issue is a matter of public health.\u003c/p>\n\u003cp>“This is about protecting kids from degenerative brain disease. It’s not unlike saying, ‘Let’s take the lead out of the paint in their home because it might rot their brain.’ Let’s not hit them in the head 500 times because it might rot their brain.”\u003c/p>\n\u003cp>But Rafter says eliminating football will affect many children from undeserved communities.\u003c/p>\n\u003cp>“[T]hey come together and experience different communities, different lifestyles through this sport,” he said.\u003c/p>\n\u003cp>Rafter said he’s received phone calls from coaches in poor neighborhoods thanking him for his efforts.\u003c/p>\n\u003cp>“I can tell you I’m getting phone calls and emails from coaches in undeserved communities of Los Angeles thanking us,” he said. “Because if these boys don’t have youth sports, they will get mixed into the wrong path.”\u003c/p>\n\u003cp>Nowinski, however, says football is not the solution to poverty.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>“I have a problem with the idea that we’re going to take kids that we already know are vulnerable, and we are going to hit them in the head a few hundred times a year, and open them up to this door of concussions and long-term [brain] disease,” he said. “It’s sort of doubling down on a problem.”\u003c/p>\n\n",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp>A \u003ca href=\"https://leginfo.legislature.ca.gov/faces/billNavClient.xhtml?bill_id=201720180AB2108\" target=\"_blank\" rel=\"noopener\">bill\u003c/a> that would have made California the first state to ban youth tackle football was shelved at the end of April, following major \u003ca href=\"https://www.ocregister.com/2018/04/27/law-to-ban-tackle-football-for-california-youth-falls-short-of-committee/\" target=\"_blank\" rel=\"noopener\">pushback\u003c/a> from parents, coaches and football fans.\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>Organizers collected more than 45,000 signatures opposing the ban, according to the \u003ca href=\"https://www.vcstar.com/story/news/2018/04/27/lawmakers-sideline-bill-ban-preteen-tackle-football/560916002/\" target=\"_blank\" rel=\"noopener\">Los Angeles Times\u003c/a>.\u003c/p>\n\u003cp>The \u003ca href=\"https://www.ocregister.com/2018/04/27/law-to-ban-tackle-football-for-california-youth-falls-short-of-committee/\" target=\"_blank\" rel=\"noopener\">Orange County Register\u003c/a> reported …\u003c/p>\n\u003cblockquote>\u003cp>(T)he backlash was swift and unyielding. Coaches and parents flooded phone lines, email boxes and talk radio, condemning the bills and questioning the science behind them. Within weeks, three of five bills were dead or left in legislative purgatory, crushed under the weight of youth football’s influence.\u003c/p>\u003c/blockquote>\n\u003caside class=\"pullquote alignright\">‘I have a problem with the idea that we’re going to take kids that we already know are vulnerable, and we are gonna hit them in the head a few hundred times a year.’ \u003ccite>Christopher Nowinski, co-founder of Boston University’s CTE Center\u003c/cite>\u003c/aside>\n\u003cp> \u003c/p>\n\u003cp>The \u003ca href=\"https://leginfo.legislature.ca.gov/faces/billNavClient.xhtml?bill_id=201720180AB2108\" target=\"_blank\" rel=\"noopener\">Safe Youth Football Act\u003c/a> would have barred children below age 12 from playing organized tackle football. The legislation was drafted in consultation with medical professionals who say the ban would protect youth from a degenerative brain disease known as \u003ca href=\"https://www.mayoclinic.org/diseases-conditions/chronic-traumatic-encephalopathy/symptoms-causes/syc-20370921\" target=\"_blank\" rel=\"noopener\">chronic traumatic encephalopathy\u003c/a>.\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>Those views were bolstered by a \u003ca href=\"https://www.bumc.bu.edu/busm/2018/04/30/youth-tackle-football-linked-to-earlier-onset-of-cognitive-and-emotional-symptoms-in-athletes-diagnosed-with-cte-after-death/\" target=\"_blank\" rel=\"noopener\">study \u003c/a>published April 30 that found playing youth tackle football may lead to earlier onset of cognitive, behavioral and emotional symptoms in later life.\u003c/p>\n\u003cp>Researchers at the Boston University School of Medicine conducted telephone interviews with the friends and relatives of 246 deceased football players and found that those who played tackle football before 12 experienced symptoms on average 13 years earlier than those who started playing at an older age.\u003c/p>\n\u003cp>Researchers took into account different styles of play and forms of protection.\u003c/p>\n\u003cp>“It took looking at 246 brains and seeing this in nearly 90 percent of them to say, ‘Hey, you know what, the research is becoming very clear, that you hit someone in the head too much, you’ll give them this disease,’ ” Christopher Nowinski, co-founder of Boston University’s \u003ca href=\"https://www.bu.edu/cte/\" target=\"_blank\" rel=\"noopener\">CTE Center\u003c/a>, said on \u003ca href=\"https://www.kqed.org/forum/2010101865041/after-california-ban-fails-controversy-persists-over-youth-tackle-football\" target=\"_blank\" rel=\"noopener\">KQED’s Forum program \u003c/a>last week.\u003c/p>\n\u003cp>Joe Rafter, the president of Southern Marin Youth Football who started a group to oppose the ban, disputes those findings and says the sport is “safer than its ever been.”\u003c/p>\u003cp>\u003c/p>\u003cp>\u003c/p>\n\u003cp>He pointed to revised coaching methods and behavior modification techniques that he argues have made the sport safer.\u003c/p>\n\u003cp>“The game that was played 40 to 50 years ago is no longer played now,” he said. “The techniques that are taught right now in youth football across America are to take the head out of the tackle.”\u003c/p>\n\u003cp>\u003cstrong>Public Health Issue\u003c/strong>\u003c/p>\n\u003cp>But Nowinski, a former football player, says changes made in the sport are not enough to make it safe for youth. The problem is not the occasional major hit to the head, he said, but the repetitive tackling, hitting and blocking.\u003c/p>\n\u003cp>“This is the best data that we are going to have for decades, because we have taken the effort to actually build the world’s largest brain bank of former football players, and we just really struggle to find football players who don’t have CTE who’ve had long careers,” he said. “Even if we diagnose concussions, a concussion happens maybe once out of every couple thousand of times a child is hit in the head. CTE risk is related to those thousands of hits, not to that concussion.”\u003c/p>\n\u003cp>Nowinski says the issue is a matter of public health.\u003c/p>\n\u003cp>“This is about protecting kids from degenerative brain disease. It’s not unlike saying, ‘Let’s take the lead out of the paint in their home because it might rot their brain.’ Let’s not hit them in the head 500 times because it might rot their brain.”\u003c/p>\n\u003cp>But Rafter says eliminating football will affect many children from undeserved communities.\u003c/p>\n\u003cp>“[T]hey come together and experience different communities, different lifestyles through this sport,” he said.\u003c/p>\n\u003cp>Rafter said he’s received phone calls from coaches in poor neighborhoods thanking him for his efforts.\u003c/p>\n\u003cp>“I can tell you I’m getting phone calls and emails from coaches in undeserved communities of Los Angeles thanking us,” he said. “Because if these boys don’t have youth sports, they will get mixed into the wrong path.”\u003c/p>\n\u003cp>Nowinski, however, says football is not the solution to poverty.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>“I have a problem with the idea that we’re going to take kids that we already know are vulnerable, and we are going to hit them in the head a few hundred times a year, and open them up to this door of concussions and long-term [brain] disease,” he said. “It’s sort of doubling down on a problem.”\u003c/p>\n\n\u003c/div>\u003c/p>",
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"content": "\u003cp class=\"big-cap-wrap danger-zone\">\u003cspan class=\"big-cap\">R\u003c/span>esearchers have been left empty-handed so far in their quest to uncover some measurable biological signal that could be used to \u003ca href=\"https://www.statnews.com/2017/02/15/brain-scan-autism-infant-diagnose/\">diagnose autism spectrum disorder\u003c/a>, leaving clinicians to identify the condition just based on a child’s behavior.\u003c/p>\n\u003cp class=\"danger-zone\">But on Wednesday, \u003ca href=\"http://stm.sciencemag.org/lookup/doi/10.1126/scitranslmed.aam9100\" target=\"_blank\" rel=\"noopener\">scientists reported\u003c/a> in the journal Science Translational Medicine that a hormone that regulates blood pressure could be one of those signposts.\u003c/p>\n\u003cp class=\"danger-zone\">They found that low concentrations of the molecule — called arginine vasopressin, or AVP — in the cerebrospinal fluid corresponded to autism-like social behavior in male monkeys, while a high AVP concentration signaled the most social animals. And they discovered similar results when looking at AVP concentrations in the cerebrospinal fluid, or CSF, of a small group of boys.\u003c/p>\n\u003cp class=\"\">“It’s really exciting work,” said Dr. Mollie Meffert, a molecular neuroscientist at Johns Hopkins, who was not involved in the study. “One of the most interesting things is the finding that the vasopressin in the CSF correlates with sociality in the macaques and in autism with children.”\u003c/p>\n\u003cp class=\"\">Meffert said if vasopressin concentrations are confirmed to correspond to \u003ca href=\"https://www.statnews.com/2016/03/31/autism-rate/\">autism\u003c/a>, they could perhaps be used to diagnose the condition and as a gauge to measure the effect of treatment candidates. And Karen Parker, the lead author of the study and associate professor of psychiatry and behavioral sciences at Stanford, said that the hormone could become a drug target if future studies show boosting its levels can assuage the social impairments of autism spectrum disorder.\u003c/p>\n\u003cp>[ad fullwidth]\u003c/p>\n\u003cp>Autism is diagnosed based on social and communication deficits and repetitive behaviors like hand-flapping or obsessions.\u003c/p>\n\u003cp>A biological diagnostic test for autism is so important, researchers say, because the earlier children start therapeutic interventions, the better the results. Without a medical test, children are typically diagnosed \u003ca href=\"https://www.cdc.gov/mmwr/volumes/67/ss/ss6706a1.htm?s_cid=ss6706a1_w\" target=\"_blank\" rel=\"noopener\">around age 4 1/2\u003c/a>, based on their behavior or a lag in their development.\u003c/p>\n\u003cp>Parker emphasized that the study’s findings are preliminary, and that the discovery of low AVP concentrations in boys with autism was based on so few samples that it needs to be replicated in a larger study. Future studies will also explore whether that association holds in adults and if levels of AVP or other hormones are associated with autism in girls and female animals.\u003c/p>\n\u003cp>Parker added that parents should not try to treat their children with vasopressin, which is used to regulate blood pressure and to treat a condition called diabetes insipidus, given that it has not been tested in autism, it’s not clear what the appropriate dose would be, and it can have \u003ca href=\"https://www.emedicinehealth.com/drug-vasopressin/article_em.htm#sideeffects\" target=\"_blank\" rel=\"noopener\">significant side effects.\u003c/a>\u003c/p>\n\u003cp>One challenge in studying the underlying biology of autism spectrum disorder is the lack of reliable rodent models of the condition. But Parker and colleagues have \u003ca href=\"http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0165401\" target=\"_blank\" rel=\"noopener\">in past studies\u003c/a> established that the natural\u003cstrong> \u003c/strong>variation of social functioning in monkeys can serve as a solid stand-in for people.\u003c/p>\n\u003cp>“They have the complex social cognition that we would care about for the control individuals, and if we can identify animals that have a deficit that’s spontaneously occurring — like humans do — that’s the value of the model,” said Parker, who is also an affiliate scientist at the California National Primate Research Center.\u003c/p>\n\u003cp>For the new study, the researchers identified 15 male rhesus macaques that exhibited low sociality, meaning they weren’t interested in playing with or grooming their fellow macaques, along with 15 highly social monkeys. They then ran tests looking for any significant differences in nine biological markers that \u003ca href=\"https://www.ncbi.nlm.nih.gov/pubmed/24462936\" target=\"_blank\" rel=\"noopener\">had been implicated\u003c/a> \u003ca href=\"https://www.ncbi.nlm.nih.gov/pubmed/26483618\" target=\"_blank\" rel=\"noopener\">in past studies\u003c/a> as possibly linked to autism, including the expression of certain genes, the products of certain signaling pathways, and the concentrations of the hormones AVP and oxytocin.\u003c/p>\n\u003cp>Only one stood out as a marker for the differences in social behavior: the concentration of AVP in the monkey’s cerebrospinal fluid, a colorless material that coats the brain and spinal cord. Notably, the levels of AVP in the blood showed no significant divergence in the two groups of monkeys.\u003c/p>\n\u003cp>To confirm their results, the researchers looked at a second group of male monkeys and again homed in on AVP concentration in the cerebrospinal fluid as being correlated to the sociality of the monkey — the lower the AVP, the less they interacted with their fellow macaques.\u003c/p>\n\u003cp>Then it was time to look at people.\u003c/p>\n\u003cp>Because obtaining cerebrospinal fluid is invasive — it requires a lumbar tap — the researchers relied on samples that had been previously collected from boys for other medical reasons, often as part of a chemotherapy treatment. They found that boys with autism spectrum disorder had lower AVP concentrations in their CSF, though with only 14 samples total — seven from control boys and seven from boys with autism spectrum disorder — the researchers could not draw firm conclusions.\u003c/p>\n\u003cp>Parker said that it’s not clear if low AVP concentrations in the cerebrospinal fluid might contribute to the social problems of autism or just be another consequence of the condition. She also added that it would likely just be one biological marker of autism, which has complicated, multifaceted causes.\u003c/p>\n\u003cp>But she said researchers have for several decades viewed vasopressin as an key influencer of social function, particularly in male animals. If AVP does indeed have some role to play in autism spectrum disorder, then its importance in males could help explain in part why \u003ca href=\"https://www.ncbi.nlm.nih.gov/pubmed/28545751\" target=\"_blank\" rel=\"noopener\">three to four times\u003c/a> more males are diagnosed with the condition than \u003ca href=\"https://www.statnews.com/2016/10/25/autism-girls-reveal-secrets/\">females\u003c/a>.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>“When people talk about autism, one of the things that feels a bit neglected is that the prevalence is very male-biased,” Parker said. “So if you’re thinking about disease mechanisms, it would be really interesting to think about the biology that’s informative to either male or female functioning.”\u003c/p>\n\n",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003cp class=\"big-cap-wrap danger-zone\">\u003cspan class=\"big-cap\">R\u003c/span>esearchers have been left empty-handed so far in their quest to uncover some measurable biological signal that could be used to \u003ca href=\"https://www.statnews.com/2017/02/15/brain-scan-autism-infant-diagnose/\">diagnose autism spectrum disorder\u003c/a>, leaving clinicians to identify the condition just based on a child’s behavior.\u003c/p>\n\u003cp class=\"danger-zone\">But on Wednesday, \u003ca href=\"http://stm.sciencemag.org/lookup/doi/10.1126/scitranslmed.aam9100\" target=\"_blank\" rel=\"noopener\">scientists reported\u003c/a> in the journal Science Translational Medicine that a hormone that regulates blood pressure could be one of those signposts.\u003c/p>\n\u003cp class=\"danger-zone\">They found that low concentrations of the molecule — called arginine vasopressin, or AVP — in the cerebrospinal fluid corresponded to autism-like social behavior in male monkeys, while a high AVP concentration signaled the most social animals. And they discovered similar results when looking at AVP concentrations in the cerebrospinal fluid, or CSF, of a small group of boys.\u003c/p>\n\u003cp class=\"\">“It’s really exciting work,” said Dr. Mollie Meffert, a molecular neuroscientist at Johns Hopkins, who was not involved in the study. “One of the most interesting things is the finding that the vasopressin in the CSF correlates with sociality in the macaques and in autism with children.”\u003c/p>\n\u003cp class=\"\">Meffert said if vasopressin concentrations are confirmed to correspond to \u003ca href=\"https://www.statnews.com/2016/03/31/autism-rate/\">autism\u003c/a>, they could perhaps be used to diagnose the condition and as a gauge to measure the effect of treatment candidates. And Karen Parker, the lead author of the study and associate professor of psychiatry and behavioral sciences at Stanford, said that the hormone could become a drug target if future studies show boosting its levels can assuage the social impairments of autism spectrum disorder.\u003c/p>\n\u003cp>\u003c/p>\u003c/div>",
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"content": "\u003cdiv class=\"post-body\">\u003cp>\u003c/p>\n\u003cp>Autism is diagnosed based on social and communication deficits and repetitive behaviors like hand-flapping or obsessions.\u003c/p>\n\u003cp>A biological diagnostic test for autism is so important, researchers say, because the earlier children start therapeutic interventions, the better the results. Without a medical test, children are typically diagnosed \u003ca href=\"https://www.cdc.gov/mmwr/volumes/67/ss/ss6706a1.htm?s_cid=ss6706a1_w\" target=\"_blank\" rel=\"noopener\">around age 4 1/2\u003c/a>, based on their behavior or a lag in their development.\u003c/p>\n\u003cp>Parker emphasized that the study’s findings are preliminary, and that the discovery of low AVP concentrations in boys with autism was based on so few samples that it needs to be replicated in a larger study. Future studies will also explore whether that association holds in adults and if levels of AVP or other hormones are associated with autism in girls and female animals.\u003c/p>\n\u003cp>Parker added that parents should not try to treat their children with vasopressin, which is used to regulate blood pressure and to treat a condition called diabetes insipidus, given that it has not been tested in autism, it’s not clear what the appropriate dose would be, and it can have \u003ca href=\"https://www.emedicinehealth.com/drug-vasopressin/article_em.htm#sideeffects\" target=\"_blank\" rel=\"noopener\">significant side effects.\u003c/a>\u003c/p>\n\u003cp>One challenge in studying the underlying biology of autism spectrum disorder is the lack of reliable rodent models of the condition. But Parker and colleagues have \u003ca href=\"http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0165401\" target=\"_blank\" rel=\"noopener\">in past studies\u003c/a> established that the natural\u003cstrong> \u003c/strong>variation of social functioning in monkeys can serve as a solid stand-in for people.\u003c/p>\n\u003cp>“They have the complex social cognition that we would care about for the control individuals, and if we can identify animals that have a deficit that’s spontaneously occurring — like humans do — that’s the value of the model,” said Parker, who is also an affiliate scientist at the California National Primate Research Center.\u003c/p>\n\u003cp>For the new study, the researchers identified 15 male rhesus macaques that exhibited low sociality, meaning they weren’t interested in playing with or grooming their fellow macaques, along with 15 highly social monkeys. They then ran tests looking for any significant differences in nine biological markers that \u003ca href=\"https://www.ncbi.nlm.nih.gov/pubmed/24462936\" target=\"_blank\" rel=\"noopener\">had been implicated\u003c/a> \u003ca href=\"https://www.ncbi.nlm.nih.gov/pubmed/26483618\" target=\"_blank\" rel=\"noopener\">in past studies\u003c/a> as possibly linked to autism, including the expression of certain genes, the products of certain signaling pathways, and the concentrations of the hormones AVP and oxytocin.\u003c/p>\n\u003cp>Only one stood out as a marker for the differences in social behavior: the concentration of AVP in the monkey’s cerebrospinal fluid, a colorless material that coats the brain and spinal cord. Notably, the levels of AVP in the blood showed no significant divergence in the two groups of monkeys.\u003c/p>\n\u003cp>To confirm their results, the researchers looked at a second group of male monkeys and again homed in on AVP concentration in the cerebrospinal fluid as being correlated to the sociality of the monkey — the lower the AVP, the less they interacted with their fellow macaques.\u003c/p>\n\u003cp>Then it was time to look at people.\u003c/p>\n\u003cp>Because obtaining cerebrospinal fluid is invasive — it requires a lumbar tap — the researchers relied on samples that had been previously collected from boys for other medical reasons, often as part of a chemotherapy treatment. They found that boys with autism spectrum disorder had lower AVP concentrations in their CSF, though with only 14 samples total — seven from control boys and seven from boys with autism spectrum disorder — the researchers could not draw firm conclusions.\u003c/p>\n\u003cp>Parker said that it’s not clear if low AVP concentrations in the cerebrospinal fluid might contribute to the social problems of autism or just be another consequence of the condition. She also added that it would likely just be one biological marker of autism, which has complicated, multifaceted causes.\u003c/p>\n\u003cp>But she said researchers have for several decades viewed vasopressin as an key influencer of social function, particularly in male animals. If AVP does indeed have some role to play in autism spectrum disorder, then its importance in males could help explain in part why \u003ca href=\"https://www.ncbi.nlm.nih.gov/pubmed/28545751\" target=\"_blank\" rel=\"noopener\">three to four times\u003c/a> more males are diagnosed with the condition than \u003ca href=\"https://www.statnews.com/2016/10/25/autism-girls-reveal-secrets/\">females\u003c/a>.\u003c/p>\n\u003cp>\u003c/p>\n\u003cp>“When people talk about autism, one of the things that feels a bit neglected is that the prevalence is very male-biased,” Parker said. “So if you’re thinking about disease mechanisms, it would be really interesting to think about the biology that’s informative to either male or female functioning.”\u003c/p>\n\n\u003c/div>\u003c/p>",
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"soldout": {
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"title": "SOLD OUT: Rethinking Housing in America",
"tagline": "A new future for housing",
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